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found in H. pylori gastritis. 10,11 With immunohistochemistry results with CD79a and CD138, we hypothesize that some of these crystal-laden 'histiocytes' are plasma cells that have failed to secrete immunoglobulin. Overproduction of immunoglobulins has been known to be a possible mechanism of crystal formation in CSH and has been described in plasma cell granuloma or post-transplantation plasmacytosis. 1 Although an additional or associated mechanism is uncertain at present, overproduction of immunoglobulin caused by H. pylori infection is a plausible aetiology. To the best of our knowledge, the present case is the second documented case of gastric CSH and the first case of localized CSH associated with polyclonal plasma cell proliferation probably caused by H. pylori infection.

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