No abstract

Amiloride prevented the contractile dysfunction of myocardial stunning but did not prevent arrhythmias. Hemodynamic alterations, increased myocardial blood flow and inhibition of Na+/H+ exchange or L-type Ca2+ channels alone did not account for the improved function. Inhibition of Na+/Ca2+ exchange may be the mechanism of improved postischemic function.

anterior descending coronary artery was totally occluded proximally, the left circumflex was small and free of disease, and the right coronary artery was dominant with severe disease prior to the posterior descending artery.The patient was taken to surgery for emergent repair of a ventricular septal defect. Figures 2 and 3 are from the intraoperative transesophageal echocardiogram. The patient received a ventricular septal patch; however, she expired a few days later.Mechanical complications of infarction involve disruption of necrotic cardiac tissues. Infarction expansion is the most common cause of disruption, occurring in 30% of infarctions. Interventricular septal rupture most frequently occurs within two weeks of acute infarction and is associated with first, large, and transmural infarctions, without collateral flow. Interventricular septal rupture complicating myocardial infarction (MI) is somewhat more common in women and patients

No abstract