No abstract

No abstract

No abstract

Epidemiologic studies have failed to demonstrate a consistent association between blood lipid concentrations and lipoprotein abnormalities and clinical manifestations, for example, cerebral infarction. If lipids do relate to atherosclerosis, then a possible explanation for this apparent contradiction is that risk factors for symptomatic cerebrovascular disease may not necessarily be the same as those for atherosclerosis in general, that is, the factors associated with the precipitation of the clinical event may differ from those related to the underlying process of atherosclerosis. 3Because lipids are often sampled following stroke, relations, or the lack of such, between blood lipids and stroke manifestations cannot automatically be extrap-