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A single acute dose of ethanol (6 mg/kg) has been shown in rats to result in the production of fatty liver characterized by the accumulation of triglycerides ( 1, 2 ) . Pyrazole, an inhibitor of liver alcohol dehydrogenase and the microsomal ethanol oxidizing system (MEOS) ( 3 -S ) , has been used to inhibit ethanol metabolism in experiments attempting to determine the mechanism behind hepatic triglyceride accumulation (6, 7 ) .These experiments led to somewhat conflicting results. Thus, Morgan and DiLuzio ( 7 ) (1970).

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