Aims-To investigate how young children develop the ability to undergo a visual field evaluation using regular automated perimetry. Methods-The study included 42 normal girls aged 5, 6, 7, and 8 years. Twelve locations in the 15 degrees eccentricity were tested in one eye, using an Octopus 2000R perimeter with a two level strategy. False positive and false negative catch trials were presented. The examination was performed three times in succession. Before the examination procedure, a specially designed programme was conducted for progressive familiarisation.

A rare cause ofvisual loss in AIDS patients: central retinal vein occlusion vision in the right eye to only hand movement at 3 feet, but the corrected vision in the left eye was 20/30. There was no anterior segment neovascularisation and the anterior chamber angle was grade IV open. The intraocular pressures were normal in both eyes. Funduscopic examination revealed typical finding of ischaemic CRVO, which was supported by fluorescein angiography as shown in Figure 1. Laboratory investigations at the time of the diagnosis showed haemoglobin of 7-9 g/l, haematocrit of 26-4%, WBC of 800x 106/1, and platelets of 30x 101/l. Extensive coagulation investigation including antithrombin III, heparin-cofactor II and protein S, protein C, antiphospholipid antibodies, and circulating immune complexes (CIC), did not show any abnormalities.The patient was followed without treatment for 1 year, her vision remained unchanged in the left eye, but showed some improvement in the right eye; she was able to count fingers at 1 foot. Comment This is the second case in the literature of CRVO causing visual loss in a patient with AIDS. CMV retinitis remains the most common cause of loss of vision in AIDS patients.' The prognosis and the treatment of CRVO and CMV retinitis are quite different.'" The diagnosis in our patient was suspected on funduscopic examination and supported by fluorescein angiography (Fig 1). CRVO occurred in this patient in the absence of underlying cardiovascular disease or any significant coagulation defects that can lead to thrombosis.24 As in the previously reported case, our patient developed CRVO in conjunction with the treatment ofthe anaemia. In the present case, the anaemia was treated with r- HuEPO

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