translocated into the terminal buds and subtending internodes in amounts that inhibited terminal bud growth and induced internodal abscission. During the development of these responses, the lateral buds apparently did not absorb sufficient A'-(3-nitro-l -naphthyl)phthalamic and Ar-(5 -acenaphthenyl) phthalamic acids at the dosage levels used to inhibit their subsequent growth. It is probable that any residual amounts of these compounds became inactive during this period as far as growth inhibition was concerned, since there was no evi-DEFOLIANT RESIDUES dence of inhibitory or formative effects on axillary buds that developed subsequently.