We are grateful for Ravat et al.'s interest in our article on PLA2G6-parkinsonism. 1,2,3 They reported an additional case showing some clinicoradiological diagnostic hints we highlighted, including young onset of levodopa-responsive parkinsonism, occurrence of truncal dystonia and cerebellar signs, early development of levodopa-induced dyskinesia, and presence of cerebellar atrophy as well as possible lack of iron deposition on brain MRI. 2 Their case 1 reinforces our observation that NM_003560.4(PLA2G6):c.2222G > A (p. Arg741Gln) is the most frequent variant in Indian cases of PLA2G6-parkinsonism. 2 It also adds to the hitherto scant evidence that deep brain stimulation (DBS) may be effective for managing symptoms and early complications of dopaminergic treatment in this disorder. 1,2 Further cases of PLA2G6-parkinsonism are in keeping with the main findings of our work and confirm the core phenotype depicted therein, 2-7 including another patient we identified (Table 1, Case S1). This is a 35-year-old female born to consanguineous Pakistani parents after uncomplicated pregnancy and birth. She experienced occasional falls during childhood and required crutches because of lower-limb spasticity since her teenage years. Despite a mild learning difficulty, she completed her secondary education at a normal school. At age 16, she had a psychotic FIG. 1. Sagittal T1 (A) and axial T2 (B) magnetic resonance (MR) images show mild cerebellar atrophy. 99mTc-TRODAT SPECT scan (C) reveals bilateral, asymmetric reduction of tracer uptake, more prominent on the left side, with putaminal uptake more reduced than the caudate bilaterally, consistent with the pattern seen in Parkinson's disease. [Color figure can be viewed at wileyonlinelibrary.com]