Ultra high molecular weight polyethylene (UHMWPE) fiber composite was reinforced by coating a functional layer on the surface of the UHMWPE fiber to improve interfacial adhesion between the fiber and the resin matrix. In this work, tannic acid (TA)‐Na+ composite modified UHMWPE fiber was used to improve moisture wettability and adhesion between the fiber and the resin. This approach had the advantages of being green, sustainable, lossless, low‐cost, and industrialized. The surface characteristics of UHMWPE fiber were investigated by Fourier transform infrared spectroscopy, X‐ray photoelectron spectroscopy, and scanning electron microscopy. The results show that the TA coating can improve the surface roughness, wettability, and adhesion of UHMWPE fiber, thereby improving the interface properties between fiber and resin. The tensile strength of interfacial shear strength and macro‐composites of TA coating UHMWPE fiber increased by 43.3% and 28%, respectively.

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<b><i>Background:</i></b> Long non-coding RNAs are reportedly endowed with the function of promoting or inhibiting cancer occurrence and development. The emphasis of this study was placed on the effect of lncRNA HLA complex group 11 (HCG11) on glioma progression, as well as its mechanism. <b><i>Methods:</i></b> Quantitative real-time polymerase chain reaction was utilized for detecting HCG11, miR-590-3p, and CAMD2 mRNA expression levels in glioma tissues. Western blot was adopted to examine cell adhesion molecule (CADM2) protein expression. Cell counting kit-8, BrdU, Transwell and wound healing assays were employed for investigating the malignant biological behaviors of glioma cells. RNA immunoprecipitation assay and dual-luciferase reporter assay were performed to prove the relationship between miR-590-3p and HCG11, as well as CADM2 and miR-590-3p. <b><i>Results:</i></b> HCG11 expression was lower in glioma tissues compared with that in paracancerous tissues, and its expression level was negatively correlated with WHO tumor stage. In addition, compared with in astrocyte cell line, the expression of HCG11 was lower in glioma cells. Functional experiments showed that HCG11 inhibited glioma cells migration and proliferation, while miR-590-3p facilitated these processes. Acting as a competitive endogenous RNA, HCG11 adsorbed miR-590-3p and upregulated the expression of CADM2, the target gene of miR-590-3p. <b><i>Conclusions:</i></b> HCG11 suppresses glioma cells proliferation and migration through regulating the miR-590-3p/CADM2 molecular axis.