We present extensive ultraviolet (UV) and optical photometry, as well as dense optical spectroscopy for type II Plateau (IIP) supernova SN 2016X that exploded in the nearby (∼ 15 Mpc) spiral galaxy UGC 08041. The observations span the period from 2 to 180 days after the explosion; in particular, the Swift UV data probably captured the signature of shock breakout associated with the explosion of SN 2016X. It shows very strong UV emission during the first week after explosion, with contribution of ∼ 20 -30% to the bolometric luminosity (versus 15% for normal SNe IIP). Moreover, we found that this supernova has an unusually long rise time of about 12.6 ± 0.5 days in the R band (versus ∼ 7.0 days for typical SNe IIP). The optical light curves and spectral evolution are quite similar to the fast-declining type IIP object SN 2013ej, except that SN 2016X has a relatively brighter tail. Based on the evolution of photospheric temperature as inferred from the Swif t data in the early phase, we derive that the progenitor of SN 2016X has a radius of about 930 ± 70 R ⊙ . This large-size star is expected to be a red supergiant star with an initial mass of 19 -20 M ⊙ based on the mass −− radius relation of the Galactic red supergiants, and it represents one of the most largest and massive progenitors found for SNe IIP.

Two hundred forty 1-d-old Arbor Acres commercial broiler chicks were divided into control and experimental (T1 and T2) groups that, between 8 and 42 d of age, were provided drinking water containing 0, 600, or 1,200 mg/L sodium from sodium chloride, respectively. The pulmonary hypertension syndrome (PHS) incidence and the right to total ventricle weight ratio (RV/ TV) were calculated weekly, and blood samples and lung tissues were collected weekly from 10 birds per group to evaluate the structural and hemodynamic characteristics of pulmonary vessels. Saline drinking water significantly increased the incidence of PHS and RV/TV ratios. In the T2 group the PHS mortality exhibited 2 peaks, including an acute peak from 14 to 21 d of age and a chronic peak from 35 to 42 d of age. During the acute peak of PHS mortality the blood volume (BV), filtration index (FI), and packed cell volume (PCV) increased in groups T1 and T2 when compared with the control group. During the acute peak there were no differences among groups in the ratio of wall to total area (WA/TA), medial thickness of pulmonary arteriole walls (mMTPA), the percentage of thick-walled peripheral lung vessels (%TWPV), the percentage of muscular arterioles (%MA), or the percentage of nonmuscular arterioles (%NMA) in pulmonary arterioles. During the chronic peak of PHS mortality, group T2 exhibited the highest values for %TWPV, %MA, WA/TA, and mMTPA and the lowest values for %NMA when compared with the T1 and control groups. Also during the chronic peak the groups did not differ in BV or FI, whereas PCV remained elevated above control values in groups T1 and T2. These observations indicate that hemodynamic changes related to viscous resistance to blood flow (BV, FI, PCV) predominated throughout the acute peak of PHS mortality, whereas, during the chronic stages of PHS mortality, increased vascular resistance to blood flow also was imposed by remodeling of the pulmonary vasculature.