Low back pain (LBP) is one of the leading causes of disability across the world (Hartvigsen et al., 2018;Vos et al., 2012). During the last decade alterations in pronociceptive and antinociceptive pain mechanisms have been hypothesized to contribute to the magnitude of clinical symptoms in LBP. A recent meta-analysis found facilitated temporal summation of pain (TSP) and impaired conditioned pain modulation (CPM) in subjects with LBP compared to healthy controls where