The effect of Trichoderma species in control of root-rot fungus, Fusarium solani, root-knot nematode, Meloidogyne javanica or reniform nematode, Rotylenchulus reniformis disease complex and on growth of sunflower plant was studied under greenhouse conditions. Treating two weeks old sunflower seedlings cv. Giza 1 with Trichoderma harzianum, T. viride, T. koningii, T. reesei or T. hamatum gave highly significant effect in control of fusarium-rot disease incidence and nematodes infection on sunflower roots. Infection of F. solani was highly increased in M. javanica infested soil than R. reniformis. Treatments of. Trichoderma species led to decrease Fusarium cfu counts in soil infested with either M. javanica or R. reniformis and also significantly improved the plant growth parameters. T. hamatum, T. harzianum and T. koningii gave the greatest reduction in disease incidence caused by Fusarium, M. javanica or R. reniformis infestation. Generally, there was highly significant reduction (p<0.01) in the number of fusarium-wilt disease and nematode population and increases in plant growth parameter of sunflower when treated with Trichoderma species.

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Eight Fusarium species i.e. F. subglutinans, F. solani, F. oxyspoum, F. sterilihyphosum, F. proliferatum, F. monili-forme, F. avena and F. chlamydspore isolated from mango malformed disease were tested for their ability to cause mango malformation disease and their production of moniliformin and total fumonisins (FB1 + FB2) using HPLC. A evaluated for moniliformin production, seven isolates were toxin producers, the production levels ranging from 0.51 to 8.90 µg/ml. The higher levels were produced by Fusarium subglutinans (8.51 µg/ml). Moderate concentrations of moniliformin was produced by F.moniliforme (6.90 µg/ml), F. oxysporum (6.30 µg/ml), F. proliferatum (4.10 μg/ml) and F. sterilihyphosum (1.10 μg/ml). Separation and identification of Fumonisin that was isolated from the pathogen- causing disease are made by (HPLC). A evaluated for total fumonisin production (FB1 + FB2), seven isolates were toxin producers, the production levels ranging from 0.10 to 8.30 µg/ml. The higher levels were produced by F. monili-forme (8.30 µg/ml. Moderate concentrations of fumonisin was produced by F .proliferatum (0.64 µg/ml) and F. subglutinans (0.50 µg/ml). Strong positive correlations between moniliformin and total fumonisins (FB1 + FB2) activities and malformation disease incidence by F. subglutinans, F. solani, F. oxyspoum, F. sterilihyphosum, F. proliferatum was observed