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has been investigating extensively on this phenomenon, which he named the latency relaxation (LR). It has been pointed out as the most characteristic property of the LR that the magnitude and the duration of LR are increased as the muscle is stretched within a certain limit (SANDOW, 1944; ABBOTT & RITCHIE, 1951; HILL, 1951; GULD & STEN-KNUDSEN, 1960). HILL (1951) implied that the LR was caused by the decrease in stiffness of the parallel elastic component rather than by that of the series elastic component and that the parallel elastic component might be the sarcolemma. HUXLEY (1957) proposed that Sfilaments which connected between the ends of opposite thin filaments were responsible for LR, because, if LR would occur in the same site where the interaction between actin and myosin filaments took place, one of the filaments should be continuous through a sarcomere and should bear at least a

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Ca ions were ionophoretically injected through an intracel

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