Background: Coronary collateral circulation is an alternative source of blood supply to the myocardium in coronary atherosclerotic disease. They provide adequate flow to the major epicardial branches of the coronary artery. Indicator of cardiac ischemia like stable angina pectoris may determine the presence of coronary collateral circulation.Methods and results: In this prospective observational cross sectional study, 150 patients with stable angina pectoris with or without MI (myocardial infarction) and or coronary intervention were enrolled. Presence of coronary collaterals in coronary angiogram was defined as Rentrop grade > 1. Patients were divided into two groups. Group A patients having Rentrop grade 0 and Group B patients are with collateral circulation, having Rentrop grade 1-3. Patients are compared in these groups. Total (63%) patients with stable angina were in Group B with coronary collateral circulation and only (37%) patients with stable angina pectoris were in Group A without collaterals.Conclusions: The incidence of development of coronary collaterals was significantly higher in patients with stable angina pectoris. DOI: 10.3329/uhj.v6i2.7245University Heart Journal Vol. 6, No. 2, July 2010 pp.61-64
Chikungunya virus (CHIKV) is an RNA alphavirus of the Togaviridae family that produces an acute febrile illness in humans followed by Joint pain, Itchy rash and leg swelling. This emerging virus has caused several large outbreaks in parts of Africa, Asia, and the Indian Ocean Islands and more recently in the Caribbean. This study was done from December 2015 to November 2016 on 24 confirmed Chikungunya patients with leg swelling. Peripheral vascular duplex study was done in every patients to find out the cause of leg swelling. Unilateral leg swelling 83% and Bilateral leg swelling 17%. Lower limb vascular Duplex was done in all patients. Moderate resersible lymphatic oedema in subcutaneous tissue of lower limb was found in 22 patients only. 2 patients had cellulites with mild lymphatic swelling. DVT was absent. There was mild reduction of peak systolic arterial flow in 13 patients which is secondary to pressure effect of lymphedema and leg swelling. 16 patients had non tender lymphadenopathy (>1cm in diameter), 2 had tender lymphadenopathy in inguinal region and no enlarged lymph glands was observed in rest of the 6 patients. 6 patients had neutropenia and 8 had lymphopenia. Gradual improvement of symptoms was observed with conservative treatment. Lymphedema is reversible and conservative therapy is appropriate. And Non tender lymphadenopathy does not require treatment.University Heart Journal Vol. 13, No. 1, January 2017; 13-16
Background: The subvalvular apparatus arrangementcan causeventricular torsion& deformation during cardiac cycle and interruption of papillary annular complex. As a result there was impairment of normal left ventricular strain pattern. [2] In patients with mitral stenosis, the left ventricle is small. Preservation of subvalvular apparatus thus become important in moderation of left ventricular volume in long term in patients with mitral stenosis undergoing mitral valve replacement. Methods: This cross sectional study was performed on the 32 consecutive subjects in department of cardiac surgery and cardiology of BSMMU with rheumatic mitral stenosis undergoing MVR from Jan 2013 to June 2014.Mode of surgery was determined by morphology of subvalvular apparatus which dictated the extent of the preservation. The patients were divided into two groups-Group I-With preservation of subvalvular apparatus &Group II-No preservation-where SVA was completely excised. In 2D and M Mode echocardiographic measurements:Mid-wall circumferential end systolic LV stress as calculated for ellipsoid, LV mass, the mid wall circumferential end systolic LV stress is calculated by mirsky's formula.9,10 Results: Patients with sub valvular apparatus resection (group I) had deterioration with postoperative ejection fraction in compare to group-II. Left ventricular circumferential wall stress analysis showed increased wall stress in group II after MVR. Conclusion: The increased left ventricular wall stress is responsible for poor outcome in nonpreserved group after MVR. The wall stress increases further in midterm follow up which may explain the mechanism of long term poor out come in patients with mitral stenosis.
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