No abstract

No abstract

Bipartite patella is a normal variation in ossification development. This variation is usually asymptomatic but can cause persistent and debilitating anterior knee pain with an injury. We report the case of a 56-year-old man complaining of persistent anterior left knee pain following trauma. Standard knee radiographs show a bilateral Bipartite Patella appearance, and magnetic resonance imaging shows discrete bony edema of the cancellous bone of the accessory bone and about the synchondrosis explaining the anterior knee pain, associated with a crack in the posterior compartment of the medial meniscus. Conservative care including medical treatment with non-steroidal anti-inflammatory drugs, physical therapy was used. Magnetic resonance imaging is the most valuable diagnostic tool for evaluating detailed morphologic and pathologic changes in patients with the bipartite patella.

Cerebral fat embolism is a rare and potentially fatal condition that may occur following a long bone fracture. Its characterized by respiratory, neurological, and mucocutaneous signs. Isolated severe brain syndrome remains exceptional. We report a 21-year-old male patient admitted for the cerebral manifestation of a fat embolism syndrome due to a fracture of long bone after a traffic accident injury. Neurological deterioration after a free interval was seen with generalized tonic-clonic seizures. MRI of the brain was indicated which showed numerous multifocal hyperintensities involving the deep white matter of both hemispheres producing a “starfield” appearance. This pattern of cytotoxic cerebral edema, with lesions in the white matter rather than the grey matter, is indicative of the subacute stage of fat embolism. The patient was treated with comprehensive support in the intensive care unit, he returned to normal neurological function and was discharged after 3 weeks of hospitalization.

La localisation surrénalienne isolée de la tuberculose représente moins de 2% des incidentalomes surrénaliens. C'est la cause infectieuse la plus fréquente des insuffisances surrénaliennes. Nous rapportons le cas d'un patient âgé de 53 ans, sans antécédents particuliers, qui présente un tableau d'insuffisance surrénalienne lente évoluant depuis six mois. L'examen physique n'a pas retrouvé de masse ni d'hépato-splénomégalie. La tension artérielle était à 120/60 mmHg. L'examen biologique n'a pas objectivé de syndrome inflammatoire et un taux de LDH normal. La TDM a objectivé une hypertrophie bilatérale des glandes surrénales siège de calcifications. L'intradermo réaction à la tuberculine était positive à 25mm. La recherche de BK dans les expectorations et dans les urines était négative. Le test au Quantiferon® était positif. Un traitement antibacillaire d'épreuve a été démarré avec une amélioration clinique avec prise de poids de 5kg en 12 mois. Les dosages hormonaux restent bas.