Cancer is the result of the outgrowth of a clonal population of cells from bodily tissue [1]. It has been acknowledged that early oncogenesis, also known as carcinogenesis, can be determined by cell-intrinsic features. A way to determine is by illustrating these features of both cancer cells and tumors , which are the ability to provide their growth signals, disregard to growth-inhibitory signals, eluding of apoptosis (programmed cell death), unlimited replication, sustained vascularization and malignancy, invasion of tissues through basement membranes and capillary walls. Cancer is considered to have three developmental phases: initiation, promotion, and progression. In the initiation phase, genomic changes such as point mutations, amplification and gene deletion, and rearrangements in the chromosomes within the cancer cell occur. *Author for correspondence These initiated cells expand clonally and survive promoting tumor development in the promotion phase. In the progression phase, significant tumor growth and metastasis are encompassed. Accumulation of genetic lesions also plays a role in the development of cancer. Inarguably, this phenomenon is present in the Initiation phase, but it may also be present in the promotion and progression phase. In the accumulation of genetic lesions, inactivation of tumor suppressor genes also occurs which provides the cells certain properties that are needed for tumorigenesis. Many types of research have established that both cancer cells and noncancer cells with a significant amount are the recipes for the heterocellular tumor are involved in tumor development and malignancy [2, 3].In most cases, cancer derives its name after the body part in which it emanated therefore, breast cancer is the term for the unstable growth of cells that emanated in the breast tissue. The growth results in a Re search Article

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