In this paper, the performance of a passive antiislanding method is experimentally tested for three phase (3φ) cogeneration systems. The tested method is based on determining the wavelet packet transform (WPT) high frequency sub-bands present in the d − q-axis components of instantaneous 3φ apparent powers (s d and sq), when evaluated at the point of common coupling (PCC). This passive anti-islanding method is founded based on the nature of instantaneous 3φ apparent powers that have components continuously exchanged between both sides of PCC. An islanding condition can be considered as a transient disturbance that creates non-periodic and non-stationary high frequency components in s d and sq. These frequency components can be parameterized by WPT high frequency sub-bands, which can provide accurate detection of the islanding condition. The d − q WPT-based anti-islanding method is tested for a 3φ co-generation system under various loading and power delivery conditions. Performance results reveal accurate, fast, and reliable detection and response to the islanding condition.Index Terms-3φ co-generation systems, anti-islanding protection, d − q-axis reference frame, and the wavelet packet transform.

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-Amphotericin B (AmpB) nephrotoxicity was used to assess the utility of drug-induced kidney injury (DIKI) biomarkers in an exploratory study in male cynomolgus monkeys. All animals had quantifiable levels of AmpB in plasma on days 1 and 4. There were no clinical signs of AmpB-induced toxicity in this study. The gold standard method used to confirm AmpB-induced DIKI was anatomic pathology which revealed microscopic lesions with varying grades of severity. Immunolocalization of alpha-1 microglobulin (α-1M), kidney injury molecule 1 (KIM-1), osteopontin (OPN) and neutrophil gelatinase-associated lipocalin (NGAL) proteins was evaluated in formalin-fixed, paraffin-embedded monkey kidney tissue sections. AmpB related immunoreactivities were identified in distinct nephron segments of treated monkeys including α-1M in damaged proximal tubule epithelium, KIM-1 in damaged medullary tubule epithelium, OPN mostly in the infiltrating cells of cortical tubule interstitium, and NGAL in the granular and cellular cast in dilatated cortical tubules. Variations in α-1M, KIM-1, OPN and NGAL immunolocalization appear as promising DIKI protein biomarkers when monitoring for AmpB-induced corticomedullary tubule injury in male cynomolgus monkeys.

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