No abstract

Hyperammonemic encephalopathy is a type of metabolic encephalopathy with diversified etiology. Hyperammonemia is the end result of several metabolic disorders such as congenital deficiencies of urea cycle enzymes, hepatic encephalopathy, Reye's syndrome and other toxic encephalopathies. Non-specific clinical presentation poses a great challenge in early diagnosis of this entity. Irrespective of the underlying etiology, hyperammonemia causes a distinctive pattern of brain parenchymal injury. The cingulate gyrus and insular cortex are more vulnerable to this type of toxic insult. Characteristic magnetic resonance imaging findings in combination with laboratory parameters can help to differentiate this entity from other metabolic encephalopathy and thus aiding in early diagnosis and treatment.

An uncommon case of idiopathic chondrolysis of the hip in an 11-year-old girl is reported. It was characterized by clinical presentation, laboratory tests, and imaging techniques. A differential diagnosis is discussed highlighting the radiological features for quick diagnosis.

No abstract

Suprascapular neuropathy has become increasingly recognized entity which is often overlooked and mistaken for other causes of shoulder pain and dysfunction like rotator cuff injury, shoulder impingement syndrome, cervical spondylosis and brachial plexopathy. It can be caused by a variety of anatomic and pathologic entities as the nerve courses from the brachial plexus through the suprascapular and spinoglenoid notches to innervate the supraspinatus and infraspinatus muscles. Because of the widespread availability of high-field MRI scanners now it is possible to detect the subtle perineural pathology, thereby excluding the other common causes of shoulder pain. There are scattered case reports and reviews describing suprascapular nerve (SSN) abnormalities using MRI. This article comprehensively reviews different pathologic abnormalities involving the SSN and illustrates their MR features, clinical presentation, correlation with electrophysiologic studies and surgical findings based on a review of 24 cases. We found the different clinical entities which includes trauma and a spectrum of nontraumatic etiology such as idiopathic, mass lesions compressing the nerve, intrinsic lesion like intraneural ganglion cysts of SSN, repetitive overuse, viral neuritis and chemotherapy induced neuropathy.