In this paper, we introduce NBNet, a novel framework for image denoising. Unlike previous works, we propose to tackle this challenging problem from a new perspective: noise reduction by image-adaptive projection. Specifically, we propose to train a network that can separate signal and noise by learning a set of reconstruction basis in the feature space. Subsequently, image denosing can be achieved by selecting corresponding basis of the signal subspace and projecting the input into such space. Our key insight is that projection can naturally maintain the local structure of input signal, especially for areas with low light or weak textures. Towards this end, we propose SSA, a non-local subspace attention module designed explicitly to learn the basis generation as well as the subspace projection. We further incorporate SSA with NBNet, a UNet structured network designed for end-to-end image denosing. We conduct evaluations on benchmarks, including SIDD and DND, and NBNet achieves state-of-the-art performance on PSNR and SSIM with significantly less computational cost.

Phoxim (O,O-diethyl O-(alpha-cyanobenzylideneamino) phosphorothioate) is a powerful organophosphorus pesticide with high potential for Bombyx mori larvae of silkworm exposure. However, it is possible that during the phoxim metabolism, there is generation of reactive oxygen species (ROS) and phoxim may produce oxidative stress and neurotoxicity in an intoxicated silkworm. Titanium dioxide nanoparticles (TiO2 NPs) pretreatment has been demonstrated to increase antioxidant capacity and acetylcholinesterase (AChE) activity in organisms. This study was, therefore, undertaken to determine phoxim-induced oxidative stress and neurotoxicity to determine whether phoxim intoxication alters the antioxidant system and AChE activity in the B. mori larval midgut, and to determine whether TiO2 NPs pretreatment attenuates phoxim-induced toxicity. The findings suggested that phoxim exposure decreased survival of B. mori larvae, increased malondialdehyde (MDA), carbonyl and 8-OHdG levels, and ROS accumulation in the midgut. Furthermore, phoxim significantly decreased the activities of AChE, superoxide dismutase (SOD), ascorbate peroxidase (APX), glutathione reductase (GR), glutathione-S-transferase (GST), and levels of ascorbic acid (AsA), reduced glutathione (GSH), and thiol in the midgut. TiO2 pretreatment, however, could increase AChE activity, and remove ROS via activating SOD, CAT, APX, GR, and GST, and accelerating AsA-GSH cycle, thus attenuated lipid, protein, and DNA peroxidation and improve B. mori larval survival under phoxim-induced toxicity. Moreover, this experimental system would help nanomaterials to be applied in the sericulture.

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