This corrects the article DOI: 10.1103/PhysRevLett.117.082503.

Fe-Mn-Si alloys are shape memory alloys which makeuse of the Y -E Stress-induced martensitic transformation.In this study, we report the effects of alloying additions on the shape memory effect (SME)of these alloys. It was found that the Mstemperature, the N6el temperature (TN) and the volume of stress-induced martensite govern the SME. Through the optimization of these factors we found that new alloy systems such as Fe-28Mn-6Si-5Cr. Fe-20Mn-5Si-8Cr-5Niand Fe-1 6Mn-5Si-12C~5Nialloys could exhibit good SME along with goodcorrosion resistance And it was also found that the thermomechanical treatment which improved the SME in Fe-Mn-Si base system was also effective to improve the SME of these new systems.KEYWORDS: Fe-Mn-Si alloy; shape memory ferrous alloy; shape memory effect: stress-induced Y -E martensitic transformation; Mstemperature: N6el temperature (TN): the amount of i• phase; training effect 1.

The quantum-chromodynamics (QCD) higher-order effects (next to the leading order in the running coupling constant) are calculated for the moments of structure functions in polarized deep-inelastic electroproduction. The QCD correction to the Bjorken sum rule is obtained and compared with the existing data.

No abstract

digested by collagenase/trypsin, and the digested cardiac cells were allowed to attach to the plates overnight. The attached cells included macrophages and myofibroblasts (positive for α smooth muscle actin [αSMA]) as well as other cardiac cells (Supplemental Figure 1B). Notably, cardiac myofibroblasts seemed to be more difficult than cardiac macrophages to collect using our isolation method from infarcted hearts because, as revealed by our immunohistochemical analysis, the number of cardiac myofibroblasts was the same as that of cardiac macrophages in the infarcted area (Supplemental Figure 1C). When the overnight-attached cells were cultured in 10% FBS/DMEM for more than 6 days, almost all of the cells on the plates were positive for αSMA and SM22α, 2 myofibroblast marker proteins (18, 19) (>97.9% and >93.8%, respectively) (Supplemental Figure 1, D and E), indicating that the cells were primarily composed of cardiac myofibroblasts. This is probably because only myofibroblasts were able to grow rapidly in the culture medium.Isolated cardiac macrophages and myofibroblasts were allowed to engulf fluorescently labeled apoptotic cells, and we assessed the fluorescence taken up by cardiac macrophages and administration promoted the restoration of cardiac function and morphology after MI, suggesting that MFG-E8 is a new therapeutic target for the treatment of MI.