Abstract-The Na ϩ /Ca 2ϩ exchanger (NCX) is increasingly recognized as a physiological mediator of Ca 2ϩ influx and significantly contributes to salt-sensitive hypertension. We recently reported that Ca 2ϩ influx by the NCX (1) is the primary mechanism of Ca 2ϩ entry in purinergically stimulated rat aorta smooth muscle cells and (2)
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