Symptomatic patients had more intense contrast agent enhancement in the plaque than asymptomatic patients, suggesting that contrast-enhanced carotid US may be used for plaque risk stratification.
The downregulation of β-ARs after ureter dilation, particularly for β1-AR and β3-AR in the muscular layer, suggests a potential compensatory mechanism involving increased contraction of the ureter to push urine through the obstruction. Thus, β-ARs may be a potential target for treatment of ureter obstruction.
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