Although literature frequently argues that diversity stimulates innovative work behavior, theoretical perspectives and empirical findings on this relationship remain inconsistent. Based on self-category theory, this study aims to comprehensively investigate when and how team cognitive diversity benefits or inhibits innovative work behavior. We introduced a new context of research (i.e., virtual teams) during COVID-19 and tested a moderated mediation model using a two-wave survey of 238 employees from 56 virtual teams in China. The results indicated that team cognitive diversity negatively related to knowledge sharing, which in turn inhibited innovative work behavior. In addition, openness to experience moderated the relationship between team cognitive diversity and knowledge sharing, such that cognitive diversity positively related to knowledge sharing among employees with a high openness to experience, while it negatively related to knowledge sharing among those with low openness. These findings enrich the existing literature on innovation by clarifying the mechanisms and boundary conditions of team cognitive diversity and innovative work behavior.

The association between duodenal ulcer and ambient temperatures or seasonal transitions suggests that energy metabolism is implicated in the disorder. Since antibiotic treatment is not universally effective for duodenal ulcer recovery, the theory of Helicobacter pylori colonization cannot account for all the cases. Here we hypothesize that the shunt of Krebs cycle and other pathways of energy metabolism generate oxalate, while the insoluble and rigid calcium oxalate may cause duodenal ulcer.

Glutamate-Oxaloacetate Transaminase is a biomarker for organ damage. However, the underlying mechanism is not fully known. The rise of Glutamate-Oxaloacetate Transaminase activity enhances the conversion of oxaloacetate to aspartic acid, resulting in lower levels of oxalate from breakdown of oxaloacetate. The insoluble and stiff calcium oxalate was proposed as a cause of cell senescence and death. Therefore, Glutamate-Oxaloacetate Transaminase reduces stress generated from energy metabolism, serving as a self-protecting mechanism upon organ damage.