Septic arthritis of the temporo-mandibular joint (TMJ) is a rare disease that has been reported infrequently. To the best of the authors' knowledge, only one case of bilateral TMJ septic arthritis has been reported. The contamination may result from direct extension of adjacent infection (dental or ENT), from hematogenous spread of blood-borne organisms or from direct inoculation. The most common presenting are trismus and pain, although swelling, tenderness and erythema have also been described. In addition, patients may develop fever, regional lymphadenopathy and malocclusion. Through a successively bilateral case of TMJ arthritis, without obvious portal of entry of the bacteria, we will analyze characteristics and treatment of this disease.

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Résumé -Introduction : le kératokyste odontogéne (KKO) est une des tumeurs odontogènes les plus fréquentes. Plusieurs travaux se sont intéressés à l'étude de cette lésion depuis sa première description afin de pouvoir comprendre son comportement biologique. Matériel et méthode : il s'agit d'une étude rétrospective immunohistochimique des marqueurs PCNA, Ki67 et P53 chez 40 patients opérés pour un KKO. Résultats : ces marqueurs sont retrouvés dans les couches suprabasales de l'épithélium de la paroi kystique. Il n'y a pas de différence statistiquement significative dans l'expression de ces marqueurs entre les différentes formes cliniques (sporadique, récurrent et syndromique), et il n'existe pas de corrélation entre ces marqueurs et la survenue d'une récidive. Conclusion : PCNA, Ki67 et P53 ne sont pas des marqueurs pour l'histopronostic des KKO.Abstract -Immunohistochemical study of proliferative (Ki67, PCNA) and apoptotic (P53) factors of 40 cases of odontogenic keratocysts. Introduction: the odontogenic keratocyst (OKC) is one of the most prevalent odontogenic tumours. Since its initial description, a number of studies have focused on different aspects of this lesion, attempting to explain its distinctive biological behavior. Patients and methods: a retrospective immunohistochemical studies of the expression of PCNA, Ki67 and p53 protein in 40 patients treated for OKC. Results: in the OKCs, the positivity of these markers was expressed mostly in the suprabasal layers of cystic epithelium. There is no statistically significant difference of expression between the different clinical forms (sporadic, recurrent and syndromic OKC), neither a correlation between the expression of this markers and recurrence. Conclusion: PCNA, P53 and Ki67 can not be considered as prognostic factors of OKC.

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