Actinic reticuloid

Chronic actinic dermatitis: A retrospective analysis of 44 cases referred to an Australian photobiology clinic

“…In this study, approximately three‐quarters of patch‐/photopatch‐tested patients had at least one ACD, PACD or combined ACD/PACD reaction. The reported incidence of contact dermatitis in CAD varies, with up to 75% of patients tested developing reactions to one or more allergens, such as compositae oleoresins, fragrances, lichens, colophony extracts, rubber chemicals and chromates 6,9,12,14,16,17 . A comparison of patch and photopatch test results is shown in Table 3.…”

“…Airborne compositae dermatitis, which may mimic CAD or precede the onset of photosensitivity, has been reported to occur in up to 86% of patients with CAD 6,16–18 . Although compositae may play a role in the pathogenesis of CAD, sesquiterpene lactone, the main allergenic constituent of the compositae plant family and a common ingredient in commercial skin preparations and cosmetics, has been suggested to be more important in the chronic evolution of this condition 14 .…”

“…The action spectrum for CAD has been shown to resemble that for normal sunburn, suggesting that an endogenous chromophore(s) such as DNA, RNA or a related molecule may be implicated 21 . In addition, the presence of pre‐existing widespread, often airborne, contact dermatitis to exogenous sensitizer or photosensitizer may be a predisposing factor in CAD 13,14,17 . It has been proposed that compositae allergy or photoallergy may simply be a result of the ability of airborne antigens to penetrate compromised skin 22 .…”

Cutaneous T-cell lymphoma in skin of color

“…With actinic reticuloid, it is not unusual to have a minimal erythema dose and even lesion reproduction with doses of 5 to 10 mJ. 18 Actinic reticuloid is uncommon in patients with skin of color. However, if this diagnosis is not considered in the differential diagnosis, these patients can be misdiagnosed as having MF.…”

Dermatitis actínica crónica en el mundo laboral

“…En la primera de ellas, un constituyente de la piel normal se altera y se vuelve antigénico, tras la unión fotoquímica covalente a un hapteno, dependiendo de UVA principalmente y en menor medida de UVB, durante una reacción fotoalérgica local. Una vez inducida la respuesta local, se perpetúa la respuesta inflamatoria, sin necesidad de que persista la reacción fotoalérgica previa 4 . Esta teoría explica la presencia de un fotoalérgeno, como desencadenante del cuadro.…”