Acute disseminated encephalomyelitis and poststreptococcal acute glomerulonephritis

Ito, Shuichi; Nezu, Atsuo; Matsumoto, Chizuru; Aihara, Yukoh; Yokota, Shumpei

doi:10.1016/s0387-7604(01)00403-x

Cited by 13 publications

(4 citation statements)

References 9 publications

Supporting

Mentioning

Contrasting

Unclassified

Order By: Relevance

“…While neuroimaging abnormalities in PRES are seen maximally in the white matter of the parietal and occipital regions, lesions can be more extensive, spreading into the cerebellum, frontal regions, basal ganglia, thalami, brain stem and corpus callosum, suggesting a diagnosis of demyelinating disease. 13 Co-existent cortical involvement is common, occurring in up to 94%. 1,14,15 Although abnormalities may be seen on computed tomography (CT) scan, fluidattenuated inversion recovery (FLAIR) MRI sequences are much better at showing the extent of the hyperintense lesions and the degree of cortical involvement.…”

Section: Discussionmentioning

confidence: 99%

Acute Glomerulonephritis Presenting With PRES: A Report of 4 Cases

Wirrell

Hamiwka

Grisaru

et al. 2007

Can. j. neurol. sci.

View full text Add to dashboard Cite

Objective:Posterior reversible encephalopathy syndrome (PRES) occurs most commonly in the setting of known hypertension or use of immunosuppressive agents.Design and Methods:We report four previously-well children who presented acutely with altered mentation, seizures and visual disturbances and were diagnosed with PRES.Results:Only one child had a history of gross hematuria prior to the seizure. All four were discovered to be hypertensive only after onset of their neurological symptoms, and were subsequently diagnosed with glomerulonephritis. All four had rapid resolution of neurological symptoms with adequate treatment of hypertension.Conclusions:Blood pressure must be measured promptly in all children presenting with these symptoms. If elevated, the diagnosis of PRES should be strongly considered and a workup for renal disease pursued.

show abstract

Section: Discussionmentioning

confidence: 99%

Acute Glomerulonephritis Presenting With PRES: A Report of 4 Cases

Wirrell

Hamiwka

Grisaru

et al. 2007

Can. j. neurol. sci.

View full text Add to dashboard Cite

show abstract

“…En nuestra serie se identificó microbiológicamente el desencadenante infeccioso en 3 casos: VEB, Mycoplasma y EBHGA. Estos desencadenantes han sido descritos en la bibliografía 10,26,27 . La vacunación previa con la triple vírica se consideró el desencadenante en 1 caso, y aunque en la actualidad se considera la principal causa de EMAD posvacunal es infrecuente en las grandes series recientes.…”

Section: Discussionunclassified

Encefalomielitis aguda. Experiencia de un hospital terciario español

Sánchez

Beato

Díaz

et al. 2005

Anales de Pediatría

View full text Add to dashboard Cite

“…In termini di mortalità e di danni neurologici, questi erano più bassi dopo una varicella o una rosolia che dopo un morbillo [23,24] . Infezioni batteriche come quelle da Borrelia burgdorferi, da leptospire e da streptococco ß-emolitico del gruppo A sono associate a un rischio di encefalomielite e di polinevriti [25][26][27][28][29] . La causa principale di infezione batterica coinvolta nello sviluppo di EMAD è il micoplasma [26] .…”

Section: Emad Postinfettiveunclassified

“…La causa principale di infezione batterica coinvolta nello sviluppo di EMAD è il micoplasma [26] . Grazie ai progressi nella gestione delle malattie infettive gravi, la cui diagnosi è più facile, l'EMAD nei paesi sviluppati sembra più frequente dopo infezioni non specifiche delle vie aeree superiori la cui eziologia è ancora sconosciuta [28][29][30] .…”

Section: Emad Postinfettiveunclassified