Anton‐Babinski syndrome in a child with early‐stage adrenoleukodystrophy

Trifiletti, R. R.; Syed, Ehteshamudin; Hayes-Rosen, Caroline; Parano, Enrico; Pavone, Piero

doi:10.1111/j.1468-1331.2006.01581.x

Cited by 16 publications

(6 citation statements)

References 6 publications

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“…Anton–Babinski syndrome is one of specific form of cortical blindness. The aetiologies include the following: ischemia stroke or haemorrhage involving occipital lobes; posterior leukoencephalopathy induced by chemotherapy, radiotherapy, preeclampsia, adrenoleukodystrophy, or mitochondrial encephalomyopathy, lactic acidosis, and stroke‐like episodes; obstetric haemorrhage with posterior circulation hypoperfusion; and trauma‐related optic neuropathy, bifrontal contusions, and callosal disconnection . Recovery of visual function can be expected if the underlying factor is posterior reversible leukoencephalopathy or cortical hypoperfusionis, and it is corrected quickly .…”

Section: Discussionmentioning

confidence: 99%

Anton–Babinski syndrome in an old patient: a case report and literature review

et al. 2014

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Anton-Babinski syndrome is a rare disease featuring bilateral cortical blindness and anosognosia with visual confabulation, but without dementia or any memory impairment. It has a unique neuropsychiatric presentation and should be highly suspected in those with odd visual loss and imaging evidence of occipital lobe injury. In the case discussed herein, a 90-year-old man presented with bilateral blindness, obvious anosognosia, and vivid visual confabulation, which he had had for 3 days. Brain computed tomography demonstrated recent hypodense infarctions at the bilateral occipital lobes. Thus, the patient was diagnosed with Anton-Babinski syndrome. Because of his age and the thrombolytic therapy during the golden 3 hours after ischemic stroke, the patient received aspirin therapy rather than tissue plasminogen activator or warfarin. He gradually realized he was blind during the following week, but died of pneumonia 1 month later. In the literature, it is difficult to establish awareness of blindness in patients with Anton-Babinski syndrome, but optimistically, in one report, a patient was aware of blindness within 2 weeks, without vision improvement. Our case illustrates that elderly patients with Anton-Babinski syndrome can partially recover and that 1 week is the shortest time for the establishment of awareness of blindness for sufferers without vision improvement.

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Section: Discussionmentioning

confidence: 99%

Anton–Babinski syndrome in an old patient: a case report and literature review

et al. 2014

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“…Other diseases described as causes are MELAS [8], preeclampsia [2], obstetric hemorrhage [9], trauma [10], adrenloeucodistrophy [11], hypertensive encephalopathy [12] and angiographic procedures [13].…”

Section: Review Of the Literaturementioning

confidence: 99%

Visual Anosognosia (Anton-Babinski Syndrome): Report of Two Cases Associated with Ischemic Cerebrovascular Disease

Carvajal¹,

Cárdenas²,

Pazmiño³

et al. 2012

JBBS

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Visual anosognosia or Anton-Babinski syndrome is a rare neurological condition related to cortical blindness. The patients deny their blindness and affirm adamantly that they are capable of seeing. The clinical presentation includes confabulations and sometimes confusional states. In this article we report two patients with anosognosia related to ischemic stroke in two different sets of etiology and pathogenesis. We describe the major clinical manifestations of this syndrome and review the current medical literature. Two patients were identified, a 96-year-old male with visual anosognosia secondary to a right posterior cerebral artery thrombosis, and a 56-year-old female with the same syndrome but related to central nervous system angiitis in relation with multiple sclerosis and Hashimoto's thyroiditis. Visual anosognosia or Anton-Babinski syndrome is a rare neurological condition. However the ischemic vascular cerebral disease is a frequent etiology. We believe that this is the first report of this syndrome in relation to angiitis with a clear autoimmune pathogenesis.

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“…Bilateral cortical blindness and Anton syndrome are most commonly caused by bilateral occipital lobe lesions [ 1 , 5 , 10 ]. This syndrome was also reported in a few other medical conditions such as gynecological complications (preeclampsia and obstetric hemorrhage) [ 11 ], MELAS [ 12 ], trauma [ 13 ], adrenoleukodystrophy [ 14 ], hypertensive encephalopathy [ 15 ], and angiographic procedures [ 16 ].…”

Section: Discussionmentioning

confidence: 78%

Anton’s Syndrome due to Bilateral Ischemic Occipital Lobe Strokes

Zukić

Sinanović

Zonić

et al. 2014

Case Reports in Neurological Medicine

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We present a case of a patient with Anton's syndrome (i.e., visual anosognosia with confabulations), who developed bilateral occipital lobe infarct. Bilateral occipital brain damage results in blindness, and patients start to confabulate to fill in the missing sensory input. In addition, the patient occasionally becomes agitated and talks to himself, which indicates that, besides Anton's syndrome, he might have had Charles Bonnet syndrome, characterized by both visual loss and hallucinations. Anton syndrome, is not so frequent condition and is most commonly caused by ischemic stroke. In this particular case, the patient had successive bilateral occipital ischemia as a result of massive stenoses of head and neck arteries.

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Anton‐Babinski syndrome in a child with early‐stage adrenoleukodystrophy

Cited by 16 publications

References 6 publications

Anton–Babinski syndrome in an old patient: a case report and literature review

Anton–Babinski syndrome in an old patient: a case report and literature review

Visual Anosognosia (Anton-Babinski Syndrome): Report of Two Cases Associated with Ischemic Cerebrovascular Disease

Anton’s Syndrome due to Bilateral Ischemic Occipital Lobe Strokes

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