Assessing Myocardial Bridging and Left Ventricular Configuration by 64-Slice Computed Tomography in Patients With Apical Hypertrophic Cardiomyopathy Presenting With Chest Pain

Chen, Chao-Chin; Chen, Ming-Tsung; Lei, Meng‐Huan; Hsu, Yu-Cheng; Chung, Sheng-Liang; Sung, Yen-Jen

doi:10.1097/rct.0b013e3181b66d31

Cited by 9 publications

(2 citation statements)

References 16 publications

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“…Fifty‐four (54) articles gave prevalence data on myocardial bridges identified by CCTA: (Aiyappan, Vadanika, Karpagam, Vinayagam, & Saravanan, 2015; Arjmand Shabestari, Akhlaghpoor, Tayebivaljozi, & Fattahi, 2012; Atar et al, 2007; Aydin et al, 2015; Cademartiri et al, 2008; Chen et al, 2010; Chen, Wu, Sheu, & Chang, 2009; De Rosa et al, 2008; Dogan et al, 2016; Duran et al, 2006; Eftekhar‐Vaghefi et al, 2019; Erol & Seker, 2012; Fujioka et al, 2009; Ghadri et al, 2014; Girzadas, Varga, & Dajani, 2009; Hwang et al, 2010; Jacobs, Bod, Kim, Hecht, & Srichai, 2008; Jeong et al, 2010; Jodocy et al, 2010; Johansen, Kirsch, Araoz, & Williamson, 2008; Juan, Hwang, Lee, & Meng, 2011; Jukić et al, 2017; Kantarci et al, 2006; Karabay, Yildiz, Bagirtan, Geceer, & Uysal, 2013; Kawawa et al, 2007; Kim et al, 2010; Ko, Choi, Nam, & Hur, 2008; Konen et al, 2007; Kosar, Ergun, Ozturk, & Kosar, 2009; La Grutta et al, 2011; La Grutta et al, 2009; Lazoura, Kanavou, Vassiou, Gkiokas, & Fezoulidis, 2010; Leschka et al, 2008; Li et al, 2016; Liu et al, 2016; Liu et al, 2010; Lu, Zhang, Guo, Huang, & Merges, 2008; Lubarsky, Gupta, & Hecht, 2007; Ma, Ma, Yu, Wu, & Li, 2013; Marcos‐Alberca et al, 2011; Nakaura et al, 2014; Niu, Zhang, Cao, & Leng, 2013; Pargaonkar et al, 2020; Rubinshtein et al, 2013; Sheu et al, 2011; Sirasapalli, Christopher, & Ravilla, 2018; Smettei & Abazid, 2016; Takamura et al, 2011; Türkvatan, Güray, & Altınsoy, 2013; Van Der Velde et al, 2020; Verhagen et al, …”

Section: Resultsmentioning

confidence: 99%

Myocardial bridges: A meta‐analysis

et al. 2021

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Myocardial bridges are anatomical entities characterized by myocardium covering segments of coronary arteries. In some patients, the presence of a myocardial bridge is benign and is only incidentally found on autopsy. In other patients, however, myocardial bridges can lead to compression of the coronary artery during systolic contraction and delayed diastolic relaxation, resulting in myocardial ischemia. This ischemia in turn can lead to myocardial infarction, ventricular arrhythmias and sudden cardiac death. Myocardial bridges have also been linked to an increased incidence of atherosclerosis, which has been attributed to increased shear stress and the presence of vasoactive factors. Other studies however, demonstrated the protective roles of myocardial bridges. In this study, using systematic review and a meta‐analytical approach we investigate the prevalence and morphology of myocardial bridges in both clinical imaging and cadaveric dissections. We also discuss the pathophysiology, clinical significance, and management of these anatomical entities.

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Section: Resultsmentioning

confidence: 99%

Myocardial bridges: A meta‐analysis

et al. 2021

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“…[23][24][25] T wave inversions can disappear during long term follow-up. 26 Figure 4A shows an EKG with characteristic T wave inversions in precordial leads associated with AHCM.…”

Section: Electrocardiographymentioning

confidence: 99%

Apical hypertrophic cardiomyopathy, a review of presentation, pathophysiology, diagnosis and natural course of the disease

et al. 2016

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Introduction:Apical Hypertrophic Cardiomyopathy (AHCM) is characterized by hypertrophy of the myocardium predominantly in the left ventricular (LV) apex and has now been recognized as an uncommon phenotype of hypertrophic cardiomyopathy (HCM). 1 The incidence of AHCM ranges from 3% of all HCM patients in North America to up to 15% and 16% in reports from Japan and China respectively. 2,3 We review the latest evidence on epidemiology, pathophysiology, diagnostic modalities, management and natural course of AHCM.

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Apical Hypertrophic Cardiomyopathy: Correlations Between Echocardiographic Parameters, Angiographic Left Ventricular Morphology, and Clinical Outcomes

Chen

Lei

Hsu

et al. 2011

Clinical Cardiology

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Background:Echocardiographic parameters could be implicated in the development of apical asynergy (characterized by apical sequestration or apical aneurysm) and worse cardiovascular outcome in patients with apical hypertrophic cardiomyopathy (ApHCM).Hypothesis:Echocardiographic parameters and morphological patterns of left ventriculograms are associated with cardiovascular morbidity and mortality in patients with ApHCM.Methods:We followed 47 cases with echocardiographically documented ApHCM. Echocardiographic findings of the extent and degree of hypertrophy, sustained cavity obliteration, and paradoxical diastolic jet flow were measured. All patients underwent a cardiac catheterization except for the cases whose informed consent was not acquired. The clinical manifestations were assessed and recorded by the attending physicians during 35.4 ± 23.7 months follow‐up.Results:Among the 47 patients with ApHCM, 30 patients presented as the “pure” form and 17 patients present as the “mixed” form. Seventeen of 28 patients with sustained cavity obliteration showed paradoxical flow by echocardiography. Thirty‐one underwent left ventriculograms and showed morphological abnormalities, including “ace‐of‐spades” configuration (15/31), apical sequestration (12/31), and apical aneurysm (4/31). The results demonstrated that cardiovascular morbidities occurred in 21 of 47 patients and were closely related to the presence of mixed form ApHCM, cavity obliteration, and paradoxical flow by univariate and multivariate Cox analysis. During the period of follow‐up, 4 patients (9.5%) died, and among them 3 had concomitant apical aneurysm.Conclusions:We concluded that detection of cavity obliteration and paradoxical flow and discrimination of pure form from mixed form by echocardiography, as well apical sequestration from apical aneurysm in ApHCM patients, is warranted. © 2011 Wiley Periodicals, Inc.This work was supported in part by grants from Lo‐Tung Poh‐Ai Hospital. The authors have no other funding, financial relationships, or conflicts of interest to disclose.

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Assessing Myocardial Bridging and Left Ventricular Configuration by 64-Slice Computed Tomography in Patients With Apical Hypertrophic Cardiomyopathy Presenting With Chest Pain

Cited by 9 publications

References 16 publications

Myocardial bridges: A meta‐analysis

Myocardial bridges: A meta‐analysis

Apical hypertrophic cardiomyopathy, a review of presentation, pathophysiology, diagnosis and natural course of the disease

Apical Hypertrophic Cardiomyopathy: Correlations Between Echocardiographic Parameters, Angiographic Left Ventricular Morphology, and Clinical Outcomes

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