Associating emergency room visits with first and prolonged extreme temperature event in Taiwan: A population-based cohort study

Wang, Yu Chun; Lin, Yu; Chuang, Chun Yu; Li, Ming Hsu; Chou, Chih-Hsing; Liao, Chun Hui; Sung, Fung Chang

doi:10.1016/j.scitotenv.2011.11.073

Cited by 68 publications

(54 citation statements)

References 40 publications

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“…Increased mortality, hospital admissions, and emergency room visits worldwide have been associated with vulnerable populations exposed to heat waves (Semenza et al 1999;Knowlton et al 2009;Anderson and Bell 2011). However, several studies reported that the adverse health effects of cold temperatures may be more significant than those of heat temperatures in Spain, Canada, Shanghai, and Taiwan (Lin et al 2011;Ma et al 2011;Martin et al 2012;Wang et al 2012). Mortality risk associated with low temperatures is likely underestimated when studies fail to address the prolonged effect of low temperature (Mercer 2003;Martin et al 2012).…”

Section: Introductionmentioning

confidence: 99%

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Effects of temperature on mortality in Hong Kong: a time series analysis

Chan

2014

Int J Biometeorol

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Although interest in assessing the impacts of hot temperature and mortality in Hong Kong has increased, less evidence on the effect of cold temperature on mortality is available. We examined both the effects of heat and cold temperatures on daily mortality in Hong Kong for the last decade (2002)(2003)(2004)(2005)(2006)(2007)(2008)(2009)(2010)(2011). A quasi-Poisson model combined with a distributed lag non-linear model was used to assess the non-linear and delayed effects of temperatures on cause-specific and age-specific mortality.Non-linear effects of temperature on mortality were identified. The relative risk of non-accidental mortality associated with cold temperature (11.1°C, 1 st percentile of temperature) relative to 19.4°C(25 th percentile of temperature) was 1.17 (95% confidence interval (CI): 1.04, 1.29) for lags 0-13. The relative risk of non-accidental mortality associated with high temperature (31.5°C, 99 th percentile of temperature) relative to 27.8°C (75 th percentile of temperature) was 1.09 (95% CI: 1.03, 1.17) for lags 0-3. In Hong Kong, extreme cold and hot temperatures increased the risk of mortality. The effect of cold lasted longer and greater than that of heat. People older than 75 years were the most vulnerable group to cold temperature, while people aged 65-74 were the most vulnerable group to hot temperature.Our findings may have implications for developing intervention strategies for extreme cold and hot temperatures.

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Section: Introductionmentioning

confidence: 99%

“…Multi-city studies reported the cold effects were most significant in warm regions (Langford and Bentham 1995;Wang et al 2012) or areas with moderate winter climates (Conlon et al 2011). …”

Section: Introductionmentioning

confidence: 99%

Effects of temperature on mortality in Hong Kong: a time series analysis

Chan

2014

Int J Biometeorol

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“…108 The first annual extreme cold event was associated with increased emergency room visits for circulatory diseases in a Taiwan study. 116 In a recent Dutch study, lower temperature was a predictor of higher incidence of acute myocardial infarction and acute presentation for abdominal aortic aneurysms. 117 A limited number of studies have looked simultaneously at ambient temperature, pollution, and their interaction in increasing the risk of adverse cardiovascular outcomes.…”

Section: Temperature Pollution and Cardiovascular Riskmentioning

confidence: 96%

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confidence: 96%

New Insights Into Pollution and the Cardiovascular System

Gold

Mittleman

2013

Circulation

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Figure.Odds ratio of ischemic stroke onset for US Environmental Protection Agency categories (good and moderate) of mean ambient fine particulate matter air pollution (PM 2.5 ) levels in the 24 hours preceding stroke onset. Error bars indicate 95% confidence interval. Reproduced from Wellenius et al 5 with permission from the publisher. The increased risk was greatest within 12 to 14 hours of exposure. Update, 2010 to 2012: Cardiac Arrhythmia, Arrhythmia Precursors, and PollutionEvidence continues to be mixed for associations between pollution and documented cardiac arrhythmias or electrophysiological changes such as repolarization abnormalities that may increase the risk of arrhythmias. Even more so than studies of stroke, ascertainment of the timing of the onset of the outcome and the specific electrophysiological nature of the outcome can be challenging to ascertain without personal monitoring, which is absent in studies of administrative data sets, which often report null findings. 6 In their recent review, Link and Dockery 7 concluded that "the incremental risk of air pollution in triggering arrhythmias… is greatest for patients with underlying cardiac disease." A London study examined associations of 11 pollutants with activation of implanted cardioverter-defibrillators, finding mostly weak associations with the elevation of a number of secondary regional pollutants, the strongest of which was the nontraffic particle component sulfate. 8 Increases in premature ventricular counts, atrial fibrillation/flutter, and its precedent, P-wave complexity, were associated with increased PM 2.5 in the previous 1 to 2 hours in a Pennsylvania study of 105 middle-aged healthy nonsmokers with 24-hour ECG and personal particle monitoring.9,10 Ghio et al 11 presented a case report of new-onset atrial fibrillation that occurred 20 minutes into a controlled exposure to concentrated ambient particles and resolved within 2 hours with no sequella. In studies of vulnerable populations who have personal electrophysiological monitoring, ozone has also been considered a risk factor for arrhythmias. An increase in the maximum ozone level predicted increases in bradycardia and apnea in high-risk infants on home monitors. 12Controlled human exposure to 1-hour exposures of diesel exhaust did not influence heart rhythm or variability in a UK study.13 Increased spatial dispersion of myocardial repolarization, but not T-wave alternans, was seen after controlled human exposures to concentrated ambient particles, ozone, or a combination of the 2 exposures.14 Increases in long-term estimated residential PM 2.5 predicted increased odds of QT prolongation without overt ventricular abnormalities in the Multi-Ethnic Study of Atherosclerosis (MESA). 15 Two recent studies (the Table) also suggest acute effects of O 3 on repolarization abnormalities in sensitive subjects. 16,17 Update, 2010 to 2012: Cardiovascular Effects of the Coarse (PM 10-2.5 ) or Ultrafine (<0.1 μm) Fractions of PM 10 Findings on cardiovascular health effects of coarse or ultr...

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“…Many previous and recent studies have shown that cold and warm ambient temperatures are relevant to increased JOHE, Summer 2013; 2 (3) risk of cardiovascular diseases and respiratory morbidity (6,(9)(10)(11)(12)(13)(14)(15)(16)(17)(18)(19)(20)(21)(22)(23). Moreover, in many countries, a survey of hospitalization and emergency data has shown that ambient temperature variations affect the number and type of hospitalized patients (24)(25)(26)(27)(28). More studies on the effects of ambient temperature on cardiovascular and respiratory diseases morbidity have been conducted in developed countries that have cold weather such as North American and European counties (4).…”

Section: Introductionmentioning

confidence: 99%

Impact of diurnal temperature variations on hospitalization due to cardiovascular and respiratory diseases in Rafsanjan, Iran, in 2008-2013

Hassanlouei

Rezaeian

Sheikhfathollahi

et al. 2013

JOHE

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Received: December 2014, Accepted: February 2015 Background: Changes in global temperature and its health effects have recently been under much investigated. Consequently, these changes have affected mortality and morbidity rates which are important public health indices. The present study examines the effect of mean temperature on the frequency of hospitalization due to cardiovascular and respiratory diseases in Ali Ibn Abi Talib Hospital of Rafsanjan, Iran. Materials and Methods:In this ecological study, data on hospitalization were gathered from electronic systems of hospitals and the data from the weather organizations were extracted from the weather station. All patients not living in Rafsanjan were excluded. Data were entered into SPSS and SAS software and the association between mean temperature and hospitalization frequency were analyzed using Poisson regression models. Results: A negative significant association was found between mean temperature and mean number of hospitalizations due to cardiovascular and respiratory diseases. By each 1 °C decrease in mean temperature, the mean number of hospitalizations due to cardiovascular and respiratory diseases increased by 1% (95% CI; 0.63-1.26%) and 4.33% (95% CI; 4-4.64%), respectively. For cardiovascular diseases, there was no significant difference in mean number of admissions based on different age groups. However, in the 0-4-year age group, the mean number of hospitalizations due to respiratory diseases was significantly higher than the other age groups. For both cardiovascular and respiratory diseases, there was no significant difference between females and males in terms of the mean number of hospitalizations. Conclusions: Reduction in mean temperature, in the present study, increased the mean number of hospitalizations due to cardiovascular and respiratory diseases and it was stronger for respiratory diseases. According to these findings, people who are at risk of cardiovascular and respiratory diseases should be forbidden from exposure to extremely cold temperatures.

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Associating emergency room visits with first and prolonged extreme temperature event in Taiwan: A population-based cohort study

Cited by 68 publications

References 40 publications

Effects of temperature on mortality in Hong Kong: a time series analysis

Effects of temperature on mortality in Hong Kong: a time series analysis

New Insights Into Pollution and the Cardiovascular System

Impact of diurnal temperature variations on hospitalization due to cardiovascular and respiratory diseases in Rafsanjan, Iran, in 2008-2013

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