Association between PTPN22 and endometriosis

Ammendola, Maria; Bottini, Nunzio; Pietropolli, Adalgisa; Saccucci, Patrizia; Gloria‐Bottini, F.

doi:10.1016/j.fertnstert.2007.04.008

Cited by 32 publications

(28 citation statements)

References 11 publications

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“…We recently reported an association between endometriosis and PTPN22 polymorphism (7). This prompted us to search for possible interactions between p53 and PTPN22 concerning their effect on susceptibility to endometriosis.…”

Section: Resultsmentioning

confidence: 99%

Association of p53 codon 72 polymorphism with endometriosis

Ammendola

Gloria‐Bottini

Sesti

et al. 2008

Fertility and Sterility

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Section: Resultsmentioning

confidence: 99%

Association of p53 codon 72 polymorphism with endometriosis

Ammendola

Gloria‐Bottini

Sesti

et al. 2008

Fertility and Sterility

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“…Associations were observed in Italian and Brazilian populations (39,40), whereas no association was found in a Polish population (41). This discrepancy is most likely the result of relatively small study populations.…”

mentioning

confidence: 81%

Endometriosis and autoimmune disease: association of susceptibility to moderate/severe endometriosis with CCL21 and HLA-DRB1

Sundqvist

Falconer

Seddighzadeh

et al. 2011

Fertility and Sterility

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“…Full-text articles of the remaining four were extracted, read, and determined to be included in the meta-analysis. 13-16 Ammendola et al 13 and Gloria-Bottini et al 16 provided two and six sets of genotypic datasets, respectively, and were considered two and six studies, respectively. Added to the two articles 14,15 with singular datasets, the final number of studies included in the meta-analysis was ten.…”

Section: Methodsmentioning

confidence: 99%

Association of the protein tyrosine phosphatase non-receptor 22 polymorphism (PTPN22) with endometriosis: a meta-analysis

Pabalan¹,

Jarjanazi

Christofolini

et al. 2017

Einstein (São Paulo)

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Objective To evaluate PTPN22 C1858T polymorphism and the risk of endometriosis.Methods A meta-analysis of 10 published case-control studies (from four articles), with a total sample of 971 cases and 1,181 controls, was performed. We estimated risk (odds ratio and 95% confidence intervals) of endometriosis associations with the C1858T polymorphism.Results A significant increased risk in all genetic models of the variant T allele with endometriosis (odds ratio: 3.14-5.55; p<0.00001-0.002) was found. The analysis without the study whose controls deviated from the Hardy-Weinberg equilibrium exacerbated these effects in the homozygous and recessive models (odds ratio: 7.19-9.45; p<0.00001-0.0002). In the Italian subgroup, a significant risk association was found in the homozygous and recessive models (odds ratio: 8.72-11.12; p=0.002).Conclusion The associations observed between PTPN22 (C1858T) and the risk of endometriosis suggest this polymorphism might be a useful susceptibility marker for this disease.

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Association between PTPN22 and endometriosis

Cited by 32 publications

References 11 publications

Association of p53 codon 72 polymorphism with endometriosis

Association of p53 codon 72 polymorphism with endometriosis

Endometriosis and autoimmune disease: association of susceptibility to moderate/severe endometriosis with CCL21 and HLA-DRB1

Association of the protein tyrosine phosphatase non-receptor 22 polymorphism (PTPN22) with endometriosis: a meta-analysis

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