Bacterial endocarditis complicating left ventricle to right atrium communication

Yacoub, Magdi H.; Mansur, Alfredo José; Towers, M; Westbury, Harry

doi:10.1016/0007-0971(72)90010-1

Cited by 9 publications

(5 citation statements)

References 6 publications

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“…Conversely, Yacoub et al [92] suggested that all LV-RA defects be repaired, regardless of their size to preclude infective endocarditis. Congenital and acquired LV-RA shunts have traditionally been corrected surgically.…”

Section: Treatmentmentioning

confidence: 99%

Gerbode defect: A comprehensive review of its history, anatomy, embryology, pathophysiology, diagnosis, and treatment

Saker

Bahri

Montalbano

et al. 2017

Journal of the Saudi Heart Association

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The purpose of this paper is to survey the literature on Gerbode defect and provide an overview of its history, anatomy, development, pathophysiology, diagnosis, and treatment options. The available literature on this topic, including case reports, was thoroughly reviewed. Gerbode defect is defined as abnormal shunting between the left ventricle and right atrium resulting from either a congenital defect or prior cardiac insults. The pathophysiology underlying the development of Gerbode defect is a disease process that injures the atrioventricular septum and leads to the abnormal shunting of blood. Although the most prevalent cause of Gerbode defect has historically been congenital, an increasing trend towards acquired cases has recently been reported owing to improved diagnostic capabilities and a greater number of invasive cardiac procedures. In conclusion, Gerbode defect is an increasingly recognized condition that warrants further study.

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Section: Treatmentmentioning

confidence: 99%

Gerbode defect: A comprehensive review of its history, anatomy, embryology, pathophysiology, diagnosis, and treatment

Saker

Bahri

Montalbano

et al. 2017

Journal of the Saudi Heart Association

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“…Yacoub et al. 5 claims that all Gerbode defects of any size should be closed, while Toprak et al. 6 argues that asymptomatic patients with normal ventricular volumes and pressures and small shunts can be managed conservatively with close monitoring.…”

Section: Discussionmentioning

confidence: 99%

Complex Congenital Heart Disease: Gerbode Defect With a Bicuspid Aortic Valve and Coarctation of the Aorta

Quien¹,

Ryzhkov²,

Miras³

et al. 2023

CASE

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“…Much of what we know regarding the management approach is based on few individual reports and small series 8 . Yacoub et al 16 proposed that all Gerbode defects should be repaired, regardless of their size, to prevent the risk of infective endocarditis and due to the low probability of spontaneous closure.…”

Section: Discussionmentioning

confidence: 99%

Three centers experience with device closure of congenital Gerbode‐type perimembranous ventricular septal defects

Haddad

Boudjemline

Combes

et al. 2022

Journal of Cardiac Surgery

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Objectives: We aim to evaluate our experience with interventional closure of Gerbode-type perimembranous ventricular septal defects (pmVSDs). Methods: We performed three-center retrospective data review of patients with congenital indirect Gerbode-type pmVSDs treated percutaneously between August 2017 and May 2021. Standard safety and latest follow-up outcomes were assessed. Results: Ten patients (six females) were identified with a median age of 6.8 years (range: 2.5-54) and a median weight of 26.5 kg (range: 12-88). The median left ventricular defect size was 10 mm (range: 3-15.5). On baseline ultrasound, 6 patients had absent subaortic rim , 6 patients had trivial aortic regurgitation, and 3 patients had tear-drop-type (small) aortic cusp prolapse. The tricuspid regurgitation was graded II (n = 5) and III (n = 5). Five Lifetech Konar-Multifunctional occluders, four Amplatzer duct occluders II and one Amplatzer duct occluder I were implanted. The median fluoroscopy time was 10.4 min (range: 4.3-20.2). Preexisting aortic regurgitations remained identical. One new aortic regurgitation was identified before discharge and remained trivial after 48 months of follow-up. No heart block or tricuspid stenosis was observed on a median follow-up of 17 months (range: 3-48). All patients are symptom-free with complete shunt closure and significant regression or resolution of tricuspid regurgitation. Conclusions: Despite anatomical challenges, interventional closure of congenital indirect Gerbode-type pmVSD appears to be feasible, safe, and most importantly clinically effective using different commercially available devices. Amplatzer duct occluder II and Lifetech Konar-Multifunctional occluder offer interesting specifications to retrogradely target this specific defect with success.

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Bacterial endocarditis complicating left ventricle to right atrium communication

Cited by 9 publications

References 6 publications

Gerbode defect: A comprehensive review of its history, anatomy, embryology, pathophysiology, diagnosis, and treatment

Gerbode defect: A comprehensive review of its history, anatomy, embryology, pathophysiology, diagnosis, and treatment

Complex Congenital Heart Disease: Gerbode Defect With a Bicuspid Aortic Valve and Coarctation of the Aorta

Three centers experience with device closure of congenital Gerbode‐type perimembranous ventricular septal defects

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