2014
DOI: 10.1556/oh.2014.29836
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Bacterial translocation: gap in the shield

Abstract: A gastrointestinalis traktus a táplálék feldolgozásában betöltött emésztő-, illetve felszívófunkcióján túl kiemelkedő jelentőséggel bír a béllumenben lévő patogénekkel szemben, megakadályozva azok keringésbe jutását, és fontos szerepet játszik az immunhomeosztázis fenntartásában. Bakteriális transzlokáció alatt a gyomor-bél rendszer életképes baktériumainak vagy bakteriális termékeinek különböző, extraintestinalis szövetekbe jutása értendő. A kóros jelensé-get számos klinikai kórkép kapcsán azonosították. A ba… Show more

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Cited by 2 publications
(2 citation statements)
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“…Our results thus indicate that the NLR increases in association with intestinal protein loss regardless of the presence of histological lymphangiectasia. A possible explanation could be the systemic neutrophilic response as a reaction to bacterial translocation, caused by the loss of the intestinal mucosal barrier [ 33 ]. In IBD dogs, reduced enzyme expression, atrophy of the villi, diffuse infiltration in the lamina propria and in the epithelium with an increase in the number of inflammatory cells and hyperplasia of goblet cells have also been observed [ 33 ].…”
Section: Discussionmentioning
confidence: 99%
See 1 more Smart Citation
“…Our results thus indicate that the NLR increases in association with intestinal protein loss regardless of the presence of histological lymphangiectasia. A possible explanation could be the systemic neutrophilic response as a reaction to bacterial translocation, caused by the loss of the intestinal mucosal barrier [ 33 ]. In IBD dogs, reduced enzyme expression, atrophy of the villi, diffuse infiltration in the lamina propria and in the epithelium with an increase in the number of inflammatory cells and hyperplasia of goblet cells have also been observed [ 33 ].…”
Section: Discussionmentioning
confidence: 99%
“…A possible explanation could be the systemic neutrophilic response as a reaction to bacterial translocation, caused by the loss of the intestinal mucosal barrier [ 33 ]. In IBD dogs, reduced enzyme expression, atrophy of the villi, diffuse infiltration in the lamina propria and in the epithelium with an increase in the number of inflammatory cells and hyperplasia of goblet cells have also been observed [ 33 ]. At the moment, it is not possible to exclude that the same mechanisms that happen in humans are present also in dogs.…”
Section: Discussionmentioning
confidence: 99%