2019
DOI: 10.1111/jne.12715
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Both neural and global androgen receptor overexpression affect sexual dimorphism in the mouse brain

Abstract: Testosterone is the main endocrine mechanism mediating sexual differentiation of the mammalian brain, although testosterone signalling is complex and important mechanistic questions remain. Notably, the extent to which testosterone acts via androgen receptors (AR) in this process remains unknown and it is also not clear where testosterone acts in the body to produce sexual dimorphisms in neuroanatomy. To address these questions, we used a transgenic mouse model of Cre/loxPdriven AR overexpression in which AR w… Show more

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