“…Two core gene programs (GM2 and GM3) characterized “Phagocytosis-Th2”, which included gene networks involved in leukocyte migration (5.3%, FDR = 9.15E-05), osteoclast differentiation (4.5%, FDR = 0.006), receptor-mediated endocytosis (5.7%, FDR = 7.03E-04, COLEC12, MSR1, CD163), and antigen processing and presentation via MHC class II (6%, p = 1.42E-13, HLA-DMA ( Gao et al, 2020 ), HLA-DRB5, HLA-DMB, HLA-DRB4, HLA-DPB1, HLA-DRA, HLA-DRB3, HLA-DOA, HLA-DQA2 ( Lasky-Su et al, 2012 ), HLA-DRB1, HLA-DPA1). The clinical characteristics showed that the eosinophils (mean: 4, p = 0.0009) were abnormally increased in blood.…”