“…Rapid increase of portal vein circumfluence resistance may be the result of these pathological changes, and may result in an obvious decrease in PVPV and acute portal hypertension formation. It was reported that patients with acute hepatitis and fulminant hepatitis developed portal hypertension with the aggravation of hepatic inflammation degrees [21,22] . Additionally, PVD was difficult to dilate to compensate for PVPV decrease in time, so PVV significantly dropped in comparison to the control groups.…”