Controllable and fast quantum-information transfer between distant nodes in two-dimensional networks

Zhong, Zhi‐Rong

doi:10.1038/s41598-016-0016-1

Cited by 83 publications

(18 citation statements)

References 57 publications

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“…These results are included in the Supplementary Information (Supplementary Fig. 4 ) and show a ratio of the intensities of the peaks associated with the A 1 g and A 2 g modes 31 , 55 , 57 – 61 of ~0.4, consistent with an oxide-free surface 31 , 57 ; this value is unchanged for the CA.M-terminated sample exposed to the atmosphere for a month consistent with the passivating effect discussed above.…”

Section: Resultssupporting

confidence: 54%

Supramolecular networks stabilise and functionalise black phosphorus

Korolkov

Timokhin²,

Haubrichs³

et al. 2017

Nat Commun

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The limited stability of the surface of black phosphorus (BP) under atmospheric conditions is a significant constraint on the exploitation of this layered material and its few layer analogue, phosphorene, as an optoelectronic material. Here we show that supramolecular networks stabilised by hydrogen bonding can be formed on BP, and that these monolayer-thick films can passivate the BP surface and inhibit oxidation under ambient conditions. The supramolecular layers are formed by solution deposition and we use atomic force microscopy to obtain images of the BP surface and hexagonal supramolecular networks of trimesic acid and melamine cyanurate (CA.M) under ambient conditions. The CA.M network is aligned with rows of phosphorus atoms and forms large domains which passivate the BP surface for more than a month, and also provides a stable supramolecular platform for the sequential deposition of 1,2,4,5-tetrakis(4-carboxyphenyl)benzene to form supramolecular heterostructures.

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Section: Resultssupporting

confidence: 54%

Supramolecular networks stabilise and functionalise black phosphorus

Korolkov

Timokhin²,

Haubrichs³

et al. 2017

Nat Commun

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“…In the present study, patients with OXTC were older (57.69 years old), compared to patients with PTC and FTC. Many studies have indicated that age plays a unique prognostic role in thyroid cancer [ 18 , 20 , 21 ], and the present study confirmed that age was an independent risk factor for both cancer-specific and all-cause mortality. Furthermore, PSM for age, race, and sex confirmed that OXTC was associated with a better prognosis, compared to PTC and FTC.…”

Section: Discussionsupporting

confidence: 86%

Propensity score matching analysis of the prognosis for the rare oxyphilic subtype of thyroid cancer (Hurthle cell carcinoma)

Xiong

Zhao

et al. 2017

Oncotarget

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There is controversy regarding the prognosis of patients with oxyphilic thyroid cancer (OXTC). The present study compared the prognoses of OXTC, papillary thyroid cancer (PTC), and follicular thyroid cancer (FTC), in order to provide a new perspective regarding the treatment guidelines for these diseases. We evaluated data from patients with thyroid cancer who were included in the Surveillance, Epidemiology, and End Results database between 2004 and 2013. Patient mortality was evaluated using Cox proportional hazards regression analyses and Kaplan-Meier analyses with log-rank tests. The multivariate Cox regression analysis revealed that the cancer-specific survival rate for OXTC was similar to that for PTC, but higher than that for FTC. However, after propensity score matching for relevant factors, the cancer-specific survival rate for OXTC was higher than that for PTC and FTC. This unexpected result provides new implications for the treatment of patients with OXTC.

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“…Subsequently, the authors concluded that GPR84 does not modulate microglial responses in the spinal dorsal horn, because GPR84 deficiency caused no alterations in microglial number and activation determined by ionized calcium-binding adapter molecule 1 and p-p38 immunostaining, respectively. Nonetheless, microglia responded to nerve injury with prominent up-regulation of GPR84 mRNA in the neuropathic pain model [ 13 ], as well as our motor nerve injury model [ 3 ], in which circulating macrophages do not contribute to the microglial pool [ 14 – 16 ], and thereby conflicts with the conclusion that there is no functional consequence of GPR84 deficiency in microglia [ 13 ].…”

Section: Introductionmentioning

confidence: 99%

Agonists for G-protein-coupled receptor 84 (GPR84) alter cellular morphology and motility but do not induce pro-inflammatory responses in microglia

Wei

Tokizane

Konishi

et al. 2017

J Neuroinflammation

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BackgroundSeveral G-protein-coupled receptors (GPCRs) have been shown to be important signaling mediators between neurons and glia. In our previous screening for identification of nerve injury-associated GPCRs, G-protein-coupled receptor 84 (GPR84) mRNA showed the highest up-regulation by microglia after nerve injury. GPR84 is a pro-inflammatory receptor of macrophages in a neuropathic pain mouse model, yet its function in resident microglia in the central nervous system is poorly understood.MethodsWe used endogenous, natural, and surrogate agonists for GPR84 (capric acid, embelin, and 6-OAU, respectively) and examined their effect on mouse primary cultured microglia in vitro.Results6-n-Octylaminouracil (6-OAU), embelin, and capric acid rapidly induced membrane ruffling and motility in cultured microglia obtained from C57BL/6 mice, although these agonists failed to promote microglial pro-inflammatory cytokine expression. Concomitantly, 6-OAU suppressed forskolin-induced increase of cAMP in cultured microglia. Pertussis toxin, an inhibitor of Gi-coupled signaling, completely suppressed 6-OAU-induced microglial membrane ruffling and motility. In contrast, no 6-OAU-induced microglial membrane ruffling and motility was observed in microglia from DBA/2 mice, a mouse strain that does not express functional GPR84 protein due to endogenous nonsense mutation of the GPR84 gene.ConclusionsGPR84 mediated signaling causes microglial motility and membrane ruffling but does not promote pro-inflammatory responses. As GPR84 is a known receptor for medium-chain fatty acids, those released from damaged brain cells may be involved in the enhancement of microglial motility through GPR84 after neuronal injury.

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Controllable and fast quantum-information transfer between distant nodes in two-dimensional networks

Cited by 83 publications

References 57 publications

Supramolecular networks stabilise and functionalise black phosphorus

Supramolecular networks stabilise and functionalise black phosphorus

Propensity score matching analysis of the prognosis for the rare oxyphilic subtype of thyroid cancer (Hurthle cell carcinoma)

Agonists for G-protein-coupled receptor 84 (GPR84) alter cellular morphology and motility but do not induce pro-inflammatory responses in microglia

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