Disodium Hydrogen Phosphate Therapy in Lead Poisoning

Proctor, Charles D.; Kahn, Harry S.

doi:10.1097/00000441-195003000-00010

Cited by 5 publications

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“…Some of the therapeutic regimens used to increase the excretion of lead, or at the least remove some "diffusable" lead, have included: low calcium, high phosphorus diet (72), low calcium diet with or without the coadministration of NH.,Cl (72,73) or H3PO., (73), Na2HPO., (74,75), sodium or potassium iodide (76,77), sodium citrate (78), dimercaprol (2,3-dimercaptopropanol, BAL) (79-81), penicillamine (81)(82)(83), and edathamil (calcium versenate, CaNa2EDTA) (81-85).…”

Section: Environmental Health Perspectivesmentioning

confidence: 99%

“…Stankovic (115) reported the following concentrations for the normal, "slight," "moderate" and "severe" categories respectively: ALAU, < 4.5, <10, >10, >20 mg/I; for CPU, <90, <180, >180 and >700 1ug/. In small children (116), the upper normal for CPU was 75 ,ug/24 hr and for adults (117,118) (19), who also studied initial exposures. Hb remained at >13 mg/100 ml and there were no clinical symptoms.…”

Section: Ppe (Fep) Erythrocyte Protoporphyrin and Freementioning

confidence: 99%

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Indices of potential lead hazard.

Posner¹

1977

Environ Health Perspect

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This review is concerned with the concentrations of lead in human whole blood, erythrocytes, plasma, serum, soft tissues, bone, and urine. The extent to which redistribution of some of the bound lead occurs is outlined. The effects of lead on enzyme activities and on the accumulation of metabolic intermediates in the blood and urine are described. A brief section deals with the range ofsigns and symptoms that can occur and differences seen between symptomatic children and adults.

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