Abstract:Background: Stroke is a causative factor of disabilities and death. Various mechanisms involved in the cerebral ischemia-reperfusion pathophysiology, including oxidative stress along with inflammation. Aim: This research assessed the impact of oxytocin in lessening the detrimental effects of reperfusion in the cerebral ischemia/reperfusion (I/R) injury with the causal mechanisms. Materials: The cerebral ischemia-reperfusion injury was elicited by bilateral common carotid artery obstruction for 30 min followed … Show more
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