“…Many of these surviving axons are stripped of their myelin (demyelinated; Bunge, Puckett, Becerra, Marcillo, & Quencer, ; Guest, Hiester, & Bunge, ; Norenberg et al, ) likely due to the loss of oligodendrocytes. Accordingly, many have hypothesized that myelin regeneration is a plausible therapeutic target to promote signal conduction and protect axons from secondary damage (Alizadeh, Dyck, & Karimi‐Abdolrezaee, ; Myers, Bankston, Burke, Ohri, & Whittemore, ; Papastefanaki & Matsas, ; Plemel et al, ). However, there is no direct evidence that oligodendrocyte remyelination contributes to the partial spontaneous functional recovery after SCI.…”