Double product break point estimates ventilatory threshold in individuals with type 2 diabetes

Abstract

[Purpose] To verify the identification of the anaerobic threshold through the double product breakpoint (DPBP) method for individuals with type 2 diabetes. [Subjects and Methods] Nine individuals with T2D (7 females; age=63.2 ± 8.9 y) and 10 non-diabetic (ND) (7 females; age=58.3 ± 7.8 y) performed an incremental exercise test on a cycle ergometer. Heart rate (HR), blood pressure (BP) and expired gas were measured at the end of each stage. The ventilatory threshold (VT) and DPBP were considered as the exercise intensities above which an over proportional increases in VE and DP were observed in relation to increasing workload. [Results] No differences were observed between the workloads, HR and VO2 corresponding to the AT identified respectively by VT and DPBP. For the T2D, strong correlations between VT and DBPB workloads (r=0.853), HR (r=0.714), and VO2 (r=0.863) were found. These relationships were similar to those found for the control group (r=0.923; r=0.881; and r=0.863, respectively). [Conclusion] These results demonstrate that the DPBP enables for the prediction of AT and correlated well the VT in both the T2D and ND participants.

Glycemic Threshold as an Alternative Method to Identify the Anaerobic Threshold in Patients With Type 2 Diabetes

“…In addition, there are differences between individuals in these parameters. In patients with type 2 diabetes, this can be confirmed by the dispersion existing in the anaerobic threshold values in relation to the maximum cardiorespiratory capacity (i.e., %VO 2peakAT ), as well as by the variance of this parameter between studies ( Belli et al, 2007 ; Delevatti et al, 2016a ; Sousa et al, 2016 ). Thus, individuals with the same maximum condition can have their anaerobic threshold in different percentages of maximum capacity, presenting different internal loads for the same maximum percentage.…”

Peculiarities of hypoglycaemic therapy in acute coronary syndrome in patients with type 2 diabetes mellitus

“…СД приводит к функциональным и структурным изме нениям сердца независимо от гипертонии, атеро склероза коронарных артерий или любого другого сердечного заболевания, и это подтверждает диа бетическую кардиомиопатию [7]. Одна из основ ных теорий причинности диабетической кардио миопатии -диабетическая нейропатия, которая приводит к симпатической денервации, изменя ющей кровоток миокарда, что влияет на его пер фузию и приводит к отсутствию загрудинной боли [8]. Пациенты с атипичной клинической картиной реже поступают с диагнозом подтвержденного ИМ и имеют более высокий уровень внутрибольнич ной смертности, чем пациенты с жалобами на боль в груди.…”