1966
DOI: 10.1055/s-0028-1111606
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Eine einfache Methode zur Trennung tumor- und entzündungsbedingter Senkungsbeschleunigungen

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Cited by 9 publications
(6 citation statements)
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“…This process has been termed thermoinactivation [13,17,19,20,26,40], since at 37 ~ an el-lipoprotein proinhibitor [13,16,17,29,39] is converted by a specific serum-lipase A to an inhibitor which becomes irreversibly attached to the erythrocyte surface, thus abolishing the effect of agglomerins. This inhibitor complex of lysolecithin character [9,13,19,20,32] has a decisive significance in differentiating inflammatory processes from malignant tumours, the former being associated with a more intensive lipase activity than are the latter.…”
Section: Introductionmentioning
confidence: 98%
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“…This process has been termed thermoinactivation [13,17,19,20,26,40], since at 37 ~ an el-lipoprotein proinhibitor [13,16,17,29,39] is converted by a specific serum-lipase A to an inhibitor which becomes irreversibly attached to the erythrocyte surface, thus abolishing the effect of agglomerins. This inhibitor complex of lysolecithin character [9,13,19,20,32] has a decisive significance in differentiating inflammatory processes from malignant tumours, the former being associated with a more intensive lipase activity than are the latter.…”
Section: Introductionmentioning
confidence: 98%
“…Investigations by Ruhenstroth-Bauer et al [17,28,29,30,31,32,33,34,36,38,39], furthermore of Gross [9,10] have thrown light on the biochemical and pathophysiological factors accounting for an increased agglomeration of erythrocytes. As a result of investigations by Fahraeus [4,5] and by Bergenhem [1] a partial suspension stability of erythrocytes has been shown to take place at 37 ~ This inactivation has been ascribed to an inhibitor complex operating at an optimal temperature [13,15,19,26].…”
Section: Introductionmentioning
confidence: 99%
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“…In der vorliegenden Arbeit wird die W~irmestabilisierung der beschleunigten Blutk6rperchensenkung bei Patienten mit einer entziindlichen oder neoplastischen Erkrankung und ihre Abh~ingigkeit yon verschiedenen Plasmafaktoren untersucht. Unsere Befunde stimmen mit denen von Gross et al [28] darin tiberein, da6 die W~irmestabilisierung einer beschleunigten BKS im Plasma yon Patienten mit einer akuten oder chronischen Leberzellsch~idigung in der Regel geringer ist als im Plasma von Patienten mit einer entziindlichen Erkrankung ohne Leberbeteiligung (Tabelle 2, Abb. [64], damit nicht in Zusammenhang gebracht werden, da dieses trotz seines hohen Gehaltes an unverestertem Cholesterin der LCAT nicht als Substrat dient [73].…”
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