Wound Healing and Skin Physiology 1995
DOI: 10.1007/978-3-642-77882-7_26
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Endogenous Growth Factor Pathways May Regulate Epidermal Hyperplasia in Chronic Venous Wounds: Modulation by Hydrocolloid Dressings

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Cited by 2 publications
(1 citation statement)
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“…Since IL-2 directs the toxin to activated immune cells rather than to keratinocytes, a pathogenic role for T cells is indicated. The reciprocal also applies, i.e., psoriatic epidermal changes are only partly reversed by treatments (e.g., calcitriol, etretinate, or cyclosporine) that produce lesser reductions in infiltrating epidermal T cells (12,13 and Lu, L, P. Gilleaudeau, J. A. McLane, N. Heftler, M. Kamber, S. Gottlieb, J. G. Krueger, and A.…”
mentioning
confidence: 99%
“…Since IL-2 directs the toxin to activated immune cells rather than to keratinocytes, a pathogenic role for T cells is indicated. The reciprocal also applies, i.e., psoriatic epidermal changes are only partly reversed by treatments (e.g., calcitriol, etretinate, or cyclosporine) that produce lesser reductions in infiltrating epidermal T cells (12,13 and Lu, L, P. Gilleaudeau, J. A. McLane, N. Heftler, M. Kamber, S. Gottlieb, J. G. Krueger, and A.…”
mentioning
confidence: 99%