1996
DOI: 10.1016/s0140-6736(96)91347-1
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Excess zinc associated with severe progressive cholestasis in Cree and Ojibwa-Cree children

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Cited by 31 publications
(17 citation statements)
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“…5,11,12 In Wilson's disease, mutations in the gene coding for a regulatory ATPase lead to copper overload due to impairment of biliary copper excretion. In chronic cholestasis, the copper accumulation is ascribed to decreased excretion of copper by hepatocytes as a result of impaired bile flow.…”
Section: Discussionmentioning
confidence: 99%
“…5,11,12 In Wilson's disease, mutations in the gene coding for a regulatory ATPase lead to copper overload due to impairment of biliary copper excretion. In chronic cholestasis, the copper accumulation is ascribed to decreased excretion of copper by hepatocytes as a result of impaired bile flow.…”
Section: Discussionmentioning
confidence: 99%
“…Nevertheless, it was reported that hepatic Zn levels were unexpectedly increased in children with severe chronic cholestatic liver disease [88,89]. In primary biliary cirrhosis (PBC) patients, hepatic Zn levels were unaffected desspite the known disturbances in Cu metabolism in this disorder [90,91].…”
Section: Cholestatic Cirrhosis and Primary Biliary Cirrhosismentioning
confidence: 99%
“…11.1b ) is found within canaliculi on ultrastructural study in both PFIC-1 and BRIC-1 Phillips et al 1987 ;Klomp et al 2000 ;. 11.1b ) is found within canaliculi on ultrastructural study in both PFIC-1 and BRIC-1 Phillips et al 1987 ;Klomp et al 2000 ;.…”
Section: Atp8b1 Diseasementioning
confidence: 88%