Gas Embolism

Mathieu, Daniel; Tissier, Stéphanie; Boulo, Marie

doi:10.1007/1-4020-4448-8_13

Cited by 7 publications

(11 citation statements)

References 75 publications

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“…Also, animal studies have shown improvement after cerebral air embolism when HBO therapy was performed even after 48 h (10). Several authors recommend HBO therapy in all cases of cerebral arterial air embolism, whenever the patient is stable (1–3, 10, 17–19). In our case, there was a time delay of 16 h before HBO therapy due to circulatory instability, probably caused either by the physical presence of air or the subsequent inflammatory response (2).…”

Section: Discussionmentioning

confidence: 99%

“…I atrogenic air embolism as a complication of surgical and invasive procedures is a rare, but potentially lethal complication (1–8). The symptoms depend on the volume of air introduced into the vessel and the site of entrance, i.e.…”

mentioning

confidence: 99%

“…The symptoms depend on the volume of air introduced into the vessel and the site of entrance, i.e. venous or arterial (1–3, 9, 10). The awake patient with venous air embolism might develop respiratory and cardiac symptoms, such as sudden dyspnoea, pulmonary oedema, cardiac arrhythmias, transient cardiovascular collapse or cardiac arrest (1).…”

mentioning

confidence: 99%

“…venous or arterial (1–3, 9, 10). The awake patient with venous air embolism might develop respiratory and cardiac symptoms, such as sudden dyspnoea, pulmonary oedema, cardiac arrhythmias, transient cardiovascular collapse or cardiac arrest (1). If the air embolus is transmitted into the arterial circulation, either directly or through a patent foramen ovale, or through another shunt, neurological signs such as change in consciousness, confusion, motor deficits, pyramidal signs, hypertonia, seizures or coma may develop (1).…”

mentioning

confidence: 99%

“…The awake patient with venous air embolism might develop respiratory and cardiac symptoms, such as sudden dyspnoea, pulmonary oedema, cardiac arrhythmias, transient cardiovascular collapse or cardiac arrest (1). If the air embolus is transmitted into the arterial circulation, either directly or through a patent foramen ovale, or through another shunt, neurological signs such as change in consciousness, confusion, motor deficits, pyramidal signs, hypertonia, seizures or coma may develop (1). In the anaesthetized patient a venous air embolus may present as an immediate decrease in end‐tidal CO 2 , a decline in the oxygen saturation, concomitant with tachycardia, hypotension, reduced cardiac output or cardiac arrest (11–13).…”

mentioning

confidence: 99%

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Iatrogenic systemic air embolism treated with hyperbaric oxygen therapy

Jørgensen

Sørensen

Jansen

2008

Acta Anaesthesiol Scand

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Air embolism is a rare and potentially severe complication of surgical and invasive procedures. Emboli large enough to produce symptoms require immediate treatment because of the risk of 'gas lock' in the right side of the heart and subsequent circulatory failure. If air is transmitted to the arterial circulation through a shunt, it may cause cerebral emboli with neurological symptoms. We present two cases with venous air emboli and concurrent cerebral arterial emboli. Both patients were successfully treated with hyperbaric oxygen therapy.

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Section: Discussionmentioning

confidence: 99%

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confidence: 99%

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confidence: 99%

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confidence: 99%

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confidence: 99%

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Iatrogenic systemic air embolism treated with hyperbaric oxygen therapy

Jørgensen

Sørensen

Jansen

2008

Acta Anaesthesiol Scand

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Revisiting the role of hyperbaric oxygen therapy in knee injuries: Potential benefits and mechanisms

Leite

Tavares

Leite

et al. 2023

Journal Cellular Physiology

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Knee injury negatively impacts routine activities and quality of life of millions of people every year. Disruption of tendons, ligaments, and articular cartilage are major causes of knee lesions, leading to social and economic losses. Besides the attempts for an optimal recovery of knee function after surgery, the joint healing process is not always adequate given the nature of intra-articular environment. Based on that, different therapeutic methods attempt to improve healing capacity. Hyperbaric oxygen therapy (HBOT) is an innovative biophysical approach that can be used as an adjuvant treatment post-knee surgery, to potentially prevent chronic disorders that commonly follows knee injuries. Given the well-recognized role of HBOT in improving wound healing, further research is necessary to clarify the benefits of HBOT in damaged musculoskeletal tissues, especially knee disorders. Here, we review important mechanisms of action for HBOT-induced healing including the induction of angiogenesis, modulation of inflammation and extracellular matrix components, and activation of parenchyma cells-key events to restore knee function after injury. This review discusses the basic science of the healing process in knee injuries, the role of oxygen during cicatrization, and shed light on the promising actions of HBOT in treating knee disorders, such as tendon, ligament, and cartilage injuries.

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