General anesthesia for electroconvulsive therapy with Brugada electrocardiograph pattern

Tsutsumi, Yasuo M.; Tomiyama, Yoshinobu; Horikawa, Yousuke T.; Shibata, Yoko; Ohshita, Naohiro; Tanaka, Katsuya; Oshita, Shuzo

doi:10.2152/jmi.58.273

Cited by 14 publications

(11 citation statements)

References 22 publications

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“…were employed in the BS cases in this study. The intravenous anesthetic propofol was used for both the induction [27][28][29][30][31][32][33][34][35][36][37][38] and maintenance of anesthesia [29,[37][38][39][40] while midazolam was utilized for both premedication [31,34,37,38,41] and induction. [27,29,33] Only the one case in which ketamine and diazepam were used died, [42] and this was due to sedation with high-dose ketamine, which led to cardiac arrest.…”

Section: Discussionmentioning

confidence: 99%

“…However, concerns about neostigmine have led some authors to prefer sugammadex, which was used safely in two of the reported cases. [30,36] Additionally, we found that atropine was selected for use in only one case [54] and that glycopyrrolate [27,31,45,48] was more commonly applied as an anticholinergic agent.…”

Section: Dischargedmentioning

confidence: 99%

“…Our patients also received the NMBAs vecuronium, [27,29,34,35,43,45,46,54] rocuronium, [30][31][32][33]36] cisatracurium, [28,47] succinylcholine, [27,38,45] and atracurium [37,38,48] but succinylcholine is not normally used because of its adverse effects. [29] As an anticholinesterase agent, neostigmine is also avoided because it can lead to ST-segment elevation in BS patients.…”

Section: Dischargedmentioning

confidence: 99%

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Brugada syndrome and anesthesia

Öztürk¹

2013

Turk Gogus Kalp Dama

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Brugada sendromu, yapısal kalp hastalığı bulunmayanlarda, elektrokardiyografide V1-3 derivasyonlarında sağ dal bloku ile spontan ya da indüklenmiş ST-segment elevasyonu ile karakterize, ani kardiyak ölüme yol açan genetik bir hastalıktır. Brugada sendromu bulunan hastalarda anestezi uygulamasına ilişkin sınırlı sayıda deneyim mevcut olduğu için, klinik uygulamaya yönelik henüz bir uzlaşı raporu bulunmamaktadır. Bu sistematik derlemede nadir görülen bu kalp hastalığında uygulanan anestezi teknikleri ve anesteziklerin etkileri ve güvenliliği irdelendi.Anah tar söz cük ler: Anestezi; anestezik ilaç; Brugada sendromu.Brugada syndrome is a genetic disease leading to sudden cardiac death, which is characterized by a spontaneous or induced ST-segment elevation with right bundle branch block in V1-3 leads in electrocardiography in those without any structural heart disease. As there is a limited number of experiences on anesthetizing patients with Brugada syndrome, no consensus report for clinical practice has been available yet. In this systematic review, we discussed the anesthesia techniques, and the effects and safety of anesthesia applied in this rarely seen heart disease.

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Section: Discussionmentioning

confidence: 99%

Section: Dischargedmentioning

confidence: 99%

Section: Dischargedmentioning

confidence: 99%

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Brugada syndrome and anesthesia

Öztürk¹

2013

Turk Gogus Kalp Dama

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“…It had been long recognized that heat, acid and capsaicin might be sensed by peripheral nerve fibres, but we have established that this receptor is also functionally expressed in keratinocytes, and moreover, heat, capsaicin and acidic conditions induce excitation of keratinocytes (40)(41)(42). Low temperature caused keratinocyte excitation via TRP type A1 (TRPA1) (43). Mechanical stimuli are also sensed by keratinocytes (21,44).…”

Section: Future Perspective 2: Studies Of Itchy and Sensitive Skinmentioning

confidence: 99%

Frontiers in epidermal barrier homeostasis – an approach to mathematical modelling of epidermal calcium dynamics

Denda

Tsutsumi

et al. 2014

Experimental Dermatology

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Intact epidermal barrier function is crucial for survival and is associated with the presence of gradients of both calcium ion concentration and electric potential. Although many molecules, including ion channels and pumps, are known to contribute to maintenance of these gradients, the mechanisms involved in epidermal calcium ion dynamics have not been clarified. We have established that a variety of neurotransmitters and their receptors, originally found in the brain, are expressed in keratinocytes and are also associated with barrier homeostasis. Moreover, keratinocytes and neurons show some similarities of electrochemical behaviour. As mathematical modelling and computer simulation have been employed to understand electrochemical phenomena in brain science, we considered that a similar approach might be applicable to describe the dynamics of epidermal electrochemical phenomena associated with barrier homeostasis. Such methodology would also be potentially useful to address a number of difficult problems in clinical dermatology, such as ageing and itching. Although this work is at a very early stage, in this essay, we discuss the background to our approach and we present some preliminary results of simulation of barrier recovery.

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“…We recently demonstrated that the intracellular calcium response of keratinocytes to external mechanical stress was greater in differentiated cells, whereas the response to ATP was greater in undifferentiated cells (3,4). Thus, the properties of epidermal keratinocytes appear to be heterogeneous depending on their location (depth) within the epidermis, i.e., depending upon the level of keratinocyte differentiation.…”

Section: Introductionmentioning

confidence: 99%

Morphological and functional differences in coculture system of keratinocytes and dorsal-root-ganglion-derived cells depending on time of seeding

Tsutsumi

Goto

Denda

et al. 2011

Experimental Dermatology

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Previous study indicated that in a coculture system of keratinocytes and dorsal-root-ganglion-derived (DRG) cells, mechanical stimulation of keratinocytes induced ATP-mediated calcium propagation and excitation of DRG cells. Here, we examined two different coculture systems of keratinocytes and DRG cells. In one, we seeded keratinocytes first and then seeded DRG cells on the keratinocytes. In this system, nerve fibres from DRG cells passed between keratinocytes. Mechanical stimulation of keratinocytes did not induce excitation of DRG cells. In the other, we seeded both cell types together. At first, each cell type grew separately, forming cell aggregates. Then, nerve fibres grew out from the DRG cell aggregates to keratinocyte aggregates and penetrated into them. In this system, mechanical stimulation of keratinocytes induced excitation of the nerve fibres, but the excitation was not completely blocked by apyrase, an ATP-degrading enzyme. These results suggest that coculture of keratinocytes and DRG can generate a variety of structures, depending on the seeding conditions.

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General anesthesia for electroconvulsive therapy with Brugada electrocardiograph pattern

Cited by 14 publications

References 22 publications

Brugada syndrome and anesthesia

Brugada syndrome and anesthesia

Frontiers in epidermal barrier homeostasis – an approach to mathematical modelling of epidermal calcium dynamics

Morphological and functional differences in coculture system of keratinocytes and dorsal-root-ganglion-derived cells depending on time of seeding

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