Ginkgolide A enhances FoxO1 expression and reduces endoplasmic reticulum stress to mitigate osteoarthritis in mice

Yu, Heng; Liang, Jinghao; Bao, Yingying; Chen, Kaiye; Jin, Yangcan; Li, Xiang; Chen, Hao; Gou, Yong; Lu, Keyu; Lin, Zhongke

doi:10.1016/j.intimp.2024.113116

International Immunopharmacology

2024

DOI: 10.1016/j.intimp.2024.113116

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Ginkgolide A enhances FoxO1 expression and reduces endoplasmic reticulum stress to mitigate osteoarthritis in mice

Heng Yu,

Jinghao Liang,

Yingying Bao

et al.

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Triptophenolide Improves Rheumatoid Arthritis and Progression by Inducing Macrophage Toxicity

Pu,

2024

J Biochem & Molecular Tox

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To investigate the role and mechanism of triptophenolide (TRI) in resisting rheumatoid arthritis (RA). Network pharmacology analysis results suggested that TRI was related to multiple inflammation‐related signaling proteins, and possessed the stable structural configuration. In animal experiments, TRI suppressed RA in mice, inhibited tissue inflammation, and improved synovial injury. Moreover, TRI can suppress RA via multiple signaling pathways, and inhibiting pyroptosis is one of the feasible treatments for improving RA.

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Triptophenolide Improves Rheumatoid Arthritis and Progression by Inducing Macrophage Toxicity

Pu,

2024

J Biochem & Molecular Tox

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Ginkgolide A enhances FoxO1 expression and reduces endoplasmic reticulum stress to mitigate osteoarthritis in mice

Cited by 1 publication

References 46 publications

Triptophenolide Improves Rheumatoid Arthritis and Progression by Inducing Macrophage Toxicity

Triptophenolide Improves Rheumatoid Arthritis and Progression by Inducing Macrophage Toxicity

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