Glaucoma After Traumatic Angle Recession A Ten-Year Prospective Study

Kaufman, Jay H.; Tolpin, Daniel W.

doi:10.1016/s0002-9394(14)76303-2

Cited by 122 publications

(47 citation statements)

References 8 publications

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“…The incidence of glaucoma related to angle recession is reported to be 7-9% and can even be manifest after 10 years. 10 In this same series, lenticular opacities were seen in 16% of cases, and the incidence of posterior segment pathological conditions was 72%. Traumatic retinal pigmentation was found in 60% of cases and retinal tears in 24%.…”

Section: Discussionmentioning

confidence: 65%

Ophthalmological findings in elite amateur Turkish boxers

Hazar

Beyleroğlu²,

Subaşı³

et al. 2002

Br J Sports Med

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Objective: To evaluate the nature and incidence of pathological ocular conditions in boxing. Methods: A group of 20 active, elite, amateur, asymptomatic Turkish boxers were examined and compared with a control group composed of 20 age matched sportsmen who were not boxers. The boxers had been actively boxing for 5-20 years (mean 9.65), were aged 16-34 (mean 22.25), and weighed 51-91 kg (mean 73.07). They had been involved in 67-500 fights (mean 143.8), with 5-40 losses (mean 17.75). All were championship title holders at the national, European, Olympic, or World level.Results: An atrophic retinal hole was found in one boxer, which was treated with laser prophylaxis. In this series, the incidence of traumatic eye injuries was much lower than in the literature. Possible reasons are their young age, their division being mainly lightweight or middleweight, few fight losses, being elite boxers, their fighting distance being near or distant, protective methods, and racial factors. Conclusion: Boxing does not appear to be as hazardous to the eyes as previously reported, but prospective, longitudinal studies need to be carried out, including boxers of all divisions, ages, boxing experience, and level, and using various methods of fighting and protection. It should be made mandatory for all boxers to have a complete ophthalmological examination at the beginning of their careers and periodically thereafter.

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Section: Discussionmentioning

confidence: 65%

Ophthalmological findings in elite amateur Turkish boxers

Hazar

Beyleroğlu²,

Subaşı³

et al. 2002

Br J Sports Med

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“…Used the US Eye injury Registry to demonstrate that 3.39% of people go to develop angle recession glaucoma (ARG) at 6 months following blunt ocular trauma 2 . A 10 year prospective study of 31 eyes by Kaufmin andTolpin reported that 6% with angle recession will go on to develop glaucoma 3 . Recession of greater than 180 degree associated with 4-9% risk of incidence of glaucoma.…”

Section: Discussionmentioning

confidence: 99%

A Case Report on post Traumatic Angle Recession

Shanbhag¹,

Walawalkar²

2017

IOSR JDMS

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“…The incidence has been reported at 3.39% at 6 months 6 and, in cases of angle recession secondary to blunt trauma, 6%-7% at 10 years. [7][8] A recent anecdotal report indicates that glaucoma may occur as late as 23 years after blunt trauma. 9 This variation in reported incidence data may not only reflect time frame differences but also differences in specific etiology and consequent severity of the injury.…”

Section: Discussionmentioning

confidence: 99%

Traumatic glaucoma with features of unilateral pigment dispersion

Bowler¹,

Ellul²,

Gouws³

2014

Digit J Ophthalmol

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SummaryWe report a patient with traumatic glaucoma with features of unilateral pigment dispersion. This rare form of secondary glaucoma has only been reported twice previously, with both patients demonstrating angle recession, indicating associated damage to the trabecular meshwork. To our knowledge, this is the first such case reported in which angle recession was absent. Case ReportA 68-year-old white man presented at Conquest Hospital, Hastings, with a complaint of redness and discomfort in the left eye 2 days after suffering blunt trauma with the tip of a pool cue. Medical history was remarkable for myopia previously treated with radial keratotomies. On examination, the left eye had diffusely injected conjunctiva with anterior chamber cell, traumatic mydriasis, and an intraocular pressure (IOP) of 20 mm Hg, with no gonioscopic evidence of angle recession. Radial keratotomy scars were visible in both eyes. Neither eye had features of pigment dispersion. Fundus examination revealed cup:disc ratios of 0.3 in both eyes, with no disc features suggestive of glaucoma. He was treated as a case of traumatic iritis and started on a tapering course of topical 0.1% dexamethasone every 2 hours.At 4 weeks' follow-up, the left eye was found to have an IOP of 70 mm Hg and a quiet anterior chamber. Although the left eye was physiologically shorter than the right eye, it was noted to have a visibly asymmetric, deeper anterior chamber on examination. This finding was confirmed by measurements obtained from the IOLMaster (Carl Zeiss Meditec, Jena, Germany): axial length 26.44 mm in the right eye and 25.84 mm in the left eye, anterior chamber depth 3.21 mm in the right eye and 3.45 mm in the left eye. There were no features of pigment dispersion. The elevated IOP was believed to be secondary to steroid therapy, which was consequently discontinued. He was started on topical latanoprost once daily and dorzolamide/timolol twice daily. This had the effect of reducing his IOP to 18 mm Hg. At 22 weeks' follow-up, his IOP had risen to 54 mm Hg, and he was noted to have pigmented cells in the anterior chamber for which he was restarted on topical dexamethasone 3 times daily. A Humphrey visual field 24-2 test was performed, revealing a mean deviation of −12.32 dB and an inferior arcuate scotoma. His IOP increased to 67 mm Hg, and he was started on oral acetazolamide 250 mg twice daily and referred to a glaucoma specialist.On examination of the left eye by a specialist 3 days later, the IOP was 42 mm Hg, the anterior chamber was asymmetrically deep with peripheral iris concavity, and there were midperipheral iris transillumination defects without other signs of iris damage. Gonioscopy showed evidence of Sampaolesi's line and no clinical evidence of angle recession (Figures 1-2). The cup:disc ratio in the left eye was 0.65, with superior neuroretinal rim thinning.The patient was diagnosed with traumatic pigment dispersion syndrome without angle recession and was started on maximal topical IOP reduction therapy; other medications were disco...

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Glaucoma After Traumatic Angle Recession A Ten-Year Prospective Study

Cited by 122 publications

References 8 publications

Ophthalmological findings in elite amateur Turkish boxers

Ophthalmological findings in elite amateur Turkish boxers

A Case Report on post Traumatic Angle Recession

Traumatic glaucoma with features of unilateral pigment dispersion

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