Hipoplasia de la vena cava inferior asociada a una trombosis venosa profunda de los miembros inferiores

Martínez-Aguilar, E.; Medina-Maldonado, F.J.; González, A. Flórez; Varela-Casariego, C.; Gómez-Penas, M.; Aciń, Francisco

doi:10.1016/s0003-3170(08)01007-9

Cited by 4 publications

(4 citation statements)

References 10 publications

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“…The authors also quote an additional Spanish language publication that highlights agenesis of the IVC as an important factor to consider in young people who develop DVT, and states that the prevalence may be as high as 5% in this group 7. García-Fuster et al 8 quoted figures as high as 16.2% for the incidence of IVC anomalies in patients under 50 years of age with iliac thrombosis.…”

Section: Discussionmentioning

confidence: 99%

Hypoplasia of the inferior vena cava in a young man presenting with extensive deep venous thrombosis

Alderman¹,

Boisanger

Bottomley³

2015

BMJ Case Reports

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SUMMARYWe present a case of a 34-year-old man with extensive deep venous thrombosis (DVT) who was found to have multiple abnormalities of venous anatomy, notably hypoplasia of the inferior vena cava with azygous continuation. DVT has an annual incidence of around 1 in 1000 people in the UK. Many cases will have readily identifiable risk factors, such as underlying malignancy, immobility or thrombophilia, but at times, these might not be so straightforward and unexpected contributing factors may be found. Although relatively uncommon in the general population, congenital venous abnormalities have been found at higher incidence in young adults presenting with spontaneous DVT and should therefore remain a consideration when classical risk factors are absent. BACKGROUND

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Section: Discussionmentioning

confidence: 99%

Hypoplasia of the inferior vena cava in a young man presenting with extensive deep venous thrombosis

Alderman¹,

Boisanger

Bottomley³

2015

BMJ Case Reports

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“…The most frequent ones are the presence of a left IVC and the duplication of IVC present in 0.2-3% of individuals. The agenesis of the IVC has an incidence of 0.0005-1% in the general population and some recent studies seem to confirm its role as an important factor of predisposition for the development of DVT in young people, with a prevalence of 5% [11]. Some authors conclude that DVT presented in these areas are caused by interaction between alterations in the coagulation and risk factors obtained due to malformations, surgery, immobilization and pregnancy [6,9].…”

Section: Discussionmentioning

confidence: 99%

“…There is no data found in publications in reference to the duration of this anticoagulation. Some authors advocate for an indefinite anticoagulation due to the existence of a permanent risk factor of venous stasis caused by malformation [8,11]. Some publications propose an invasive endovascular therapy if the location of the anomaly has been clearly identified [8], although the tendency is to maintain anticoagulation indefinitely but further tests are necessary before commencing this treatment.…”

Section: Discussionmentioning

confidence: 99%

Hypoplasia of Infrarenal Inferior Vena Cava: An Infrequent Cause of Repetitive Deep Vein Thrombosis and its Effects

2015

J Hematol Thromb

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Congenital anomalies such as the defects of inferior vena cava (IVC) are not common, however these alterations obtained from a poor embryo development benefit venous stasis which, in consequence, will trigger a deep vein thrombosis (DVT). This diagnosis must be considered for young patients who have no evident risks of spontaneous and bilateral DVT.We introduce a clinical case of a 39 year-old male with iliofemoral DVT who presented recurring DVT after the withdrawal of anticoagulation, with no other predisposing factor.

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“…En los casos de TVP asociados a anomalías de la VCI, la mayoría de los autores opta por la anticoagulación profiláctica de por vida y se basa en el riesgo de recurrencia de la TVP. 15 En el caso que se presenta, se adoptó una conducta conservadora, que consistió en un seguimiento multidisciplinario por pediatras, hematólogos y especialistas en anomalías vasculares. Se decidió el manejo expectante con respecto al inicio de la anticoagulación dada la edad de la paciente.…”

Section: Venas Supracardinalesunclassified

Ascitis fetal como forma de presentación de agenesia de la vena cava inferior

Mercado

Liberto²,

Udaquiola

et al. 2018

Arch Argent Pediatr

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Inferior Vena Cava (IVC) is composed of three segments from different embryological origin. Its lack of fusion originates a wide spectrum of anomalies of the IVC. These malformations are present in 0.07-8.7% of the population. It is generally diagnosed as an incidental finding in adult life. It represents between 5 and 9.5% of idiopathic deep vein thrombosis in patients younger than 30 years old without associated risk factors. We present a case of a term newborn with prenatal diagnosis of ascites during the 20th week of gestation. IVC Agenesis was diagnosed with the use of abdominal angiotomography. The treatment of patients with IVC Agenesis is based on the management of its complications. Due to the increased thrombotic risk of these patients, we should consider lifelong anticoagulation. We suggest initiating it during puberty.

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Hipoplasia de la vena cava inferior asociada a una trombosis venosa profunda de los miembros inferiores

Cited by 4 publications

References 10 publications

Hypoplasia of the inferior vena cava in a young man presenting with extensive deep venous thrombosis

Hypoplasia of the inferior vena cava in a young man presenting with extensive deep venous thrombosis

Hypoplasia of Infrarenal Inferior Vena Cava: An Infrequent Cause of Repetitive Deep Vein Thrombosis and its Effects

Ascitis fetal como forma de presentación de agenesia de la vena cava inferior

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