Imagent Improves Endocardial Border Delineation, Inter‐reader Agreement, and the Accuracy of Segmental Wall Motion Assessment

Nanda, Navin C.; Kitzman, Dalane W.; Dittrich, Howard C.; Hall, Gail; Ahmad, Massood; Aurigemma, Gerard P.; Axel, Leon; Byrd, Benjamin F.; Chaudhry, Farooq A.; Cohen, Jacob; Copans, Harold; Crouse, Linda J.; Dennish, George W.; Ford, K.L.; Gillam, Linda D.; Goldman, Martin E.; Grayburn, Paul A.; Hill, David L.; Imburgia, M.; Jacobs, Larry E.; Kotler, Morris N.; Lighty, George W.; Mankad, Sunil; Pandian, Natesa G.; Panidis, John; Pérez, Julio E.; Raichlen, Joel S.; Rubenson, David; Ryan, Thomas J.; Segar, Douglas S.; Shapiro, Edward P.; Shirani, Jamshid; Sutton, Martin St. John; Stowers, Stephen A.; Decker, William Van; Villanueva, Flordeliza S.; Zoghbi, William A.

doi:10.1046/j.1540-8175.2003.03014.x

Cited by 28 publications

(19 citation statements)

References 17 publications

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“…Gender and interethnic differences in intimal thickening, as demonstrated histologically on autopsy material, indicated a strong genetic control on this process. Nanda et al [116] studied racial differences in thoracic aorta thickness among IS patients and found that black populations have a lower prevalence of extracranial atherosclerotic disease than Caucasians and this difference cannot be explained by the existing risk factors alone.…”

Section: Genetic Predisposition Affects Both Coronary and Carotid Athmentioning

confidence: 99%

Atherothrombosis and Plaque Heterology: Different Location or a Unique Disease?

Slevin¹,

Wang²,

Font³

et al. 2008

Pathobiology

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Formation of unstable plaques frequently results in atherothrombosis, the major cause for ischaemic stroke, myocardial infarction and peripheral arterial disease. Patients who have symptomatic thrombosis in one vascular bed are at increased risk of disease in other beds. However, the development of the disease in carotid, coronary and peripheral arteries may have different pathophysiology suggesting that more complex treatment protocols may have to be designed to reduce plaque development at different locations. In this review we describe the known risk factors, compare the developmental features of coronary and carotid plaque development and determine their association with end-point ischaemic events. Differences are also seen in the genetic contribution to plaque development as well as in the deregulation of gene and protein expression and cellular signal transduction activity of active cells in regions susceptible to thrombosis. Differences between carotid and coronary artery plaque development might help to explain the differences in anatomopathological appearance and risk of rupture.

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Section: Genetic Predisposition Affects Both Coronary and Carotid Athmentioning

confidence: 99%

Atherothrombosis and Plaque Heterology: Different Location or a Unique Disease?

Slevin¹,

Wang²,

Font³

et al. 2008

Pathobiology

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“…The use of intravenous contrast agents has been shown to improve the assessment of segmental wall motion and interobserver agreement by enhancing the endocardial border delineation (22,23). Because RTCE permits the analysis of both wall motion and perfusion, the injection of contrast agents during RTCE should also assist in detecting WMA.…”

Section: Feasibility Of Mpi Image Interpretationmentioning

confidence: 99%

Safety of dobutamine stress real-time myocardial contrast echocardiography

Tsutsui

Elhendy

Xie

et al. 2005

Journal of the American College of Cardiology

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“…A shell encapsulating the gas bubble is essential for the longevity. Proposed shell materials include protein (Albunex Ò [8] and Optison [9]), lipid (SonoVue Ò [10], Definity TM [11] and Imagent Ò [12]) or surfactants (ST68 [13]) and the gas can be perfluorocarbon [9][10][11][12][13], sulfur hexafluoride [10,13], air [14,15] or some other inert gas or gas mixture.…”

Section: Introductionmentioning

confidence: 99%