Intermittent Positive Pressure Ventilation with either Positive End-Expiratory Pressure or High Frequency Jet Ventilation (HFJV), or HFJV Alone in Human Acute Respiratory Failure

Brichant, Jean-François; Rouby, Jean-Jacques; Viars, P.

doi:10.1213/00000539-198611000-00007

Cited by 17 publications

(8 citation statements)

References 7 publications

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“…During CHFVO there was a non-signific'ant trend towards desaturation and lower Pao, values. The results here are in accordance with those of critically ill patients with CHFV (5). The authors concluded that arterial oxygenation is mainly dependent on mean Paw, in spite of the method employed to increase Paw (5).…”

Section: Cppvsupporting

confidence: 89%

“…CHFV CHFVO HR beatlmin 94f 12 89f 10 97f11 cation of CPAP, and the effect was also quickly lost after the removal of CPAP (1 1). Enhanced CO, elimination with CHFV, as compared to CPPV, has been reported (5,12). However, in the present study, no difference in CO, washout was seen between CHFV and CPPV due to the protocol; nor were there any significant differences in CO,-washout between either CHFVO and CPPV or CHFVO and CHFV.…”

Section: Cppvcontrasting

confidence: 74%

“…The pressure level in the airways, not the waveform producing it, is regarded as the major determinant of the cardiodepressive effects. Mean Paw is considered to be the principal influence on cardiac output during CHFV (5). It has also been shown that the haemodynamic effects of positive endexpiratory pressure in dogs are similar during both conventional and high frequency ventilation (15).…”

Section: Cppvmentioning

confidence: 99%

“…El-Baz et al have introduced a method of combining the beneficial effects of the two ventilation modes (3). Various applications of combining HFV with the more conventional modes of ventilation provide interesting results for treating severe acute pulmonary failure (1,4,5). ' In the present study, HFV was applied in an airway pressure release ventilation (APRV) (6) style, so that the mean airway pressure (Paw) curve would resemble APRV.…”

mentioning

confidence: 95%

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The effect of combined high frequency ventilation with and without continuous positive airway pressure in experimental lung injury

Jousela

Måkelãinen

Linko

1992

Acta Anaesthesiol Scand

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Combined high frequency ventilation (CHFV) with 8 mmHg (1.0 kPa) continuous positive airway pressure (CPAP) and without CPAP (CHFV0) were compared to each other, and to continuous positive pressure ventilation (CPPV) with 8 mmHg (1.0 kPa) CPAP in pigs with oleic acid induced lung injury. The respiratory rate was 15 min-1 and the high frequency (HF) rate 360 min-1. Arterial carbon dioxide tension (PaCO2) was adjusted to 5 kPa and 25% oxygen was used. After CHFV, CPAP was briefly discontinued to allow the establishment of CHFV0 in order to examine the cardiovascular and pulmonary effects of combined high frequency ventilation alone. Mean arterial oxygen tension (PaO2) was 15.8 +/- 3.9 kPa during CPPV, 15.5 + 3.2 kPa during CHFV and 13.2 +/- 5.1 kPa during CHFV0 (ns). The peak airway pressure and the pericardiac pressure were lowest during CHFV0. CHFV provoked significant cardiovascular depression (mean arterial pressure, stroke index, left and right ventricle stroke work index). When compared to CPPV, a non-significant trend towards improved cardiovascular function was found during CHFV0. With similar mean airway pressures (during CHFV0) or the same CPAP (during CHFV) as during CPPV, no further improvement in oxygenation due to HF waves was found. Airway pressure was the major factor causing alterations in cardiovascular function, not the ventilation technique.

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Section: Cppvsupporting

confidence: 89%

Section: Cppvcontrasting

confidence: 74%

Section: Cppvmentioning

confidence: 99%

mentioning

confidence: 95%

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The effect of combined high frequency ventilation with and without continuous positive airway pressure in experimental lung injury

Jousela

Måkelãinen

Linko

1992

Acta Anaesthesiol Scand

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“…Concerning high frequency, however, presently available studies did not show clear benefits as compared with conventional ventilation [14,50,54]. Apart from specific indications (e.g.…”

Section: Respirator Therapymentioning

confidence: 99%

Prevention and therapy of the adult respiratory distress syndrome

Temmesfeld-Wollbrück

Walmrath

Grimminger

et al. 1995

Lung

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The complex pathophysiology of adult respiratory distress syndrome (ARDS) makes preventive and therapeutic concepts difficult. Ample experimental evidence indicates that ARDS can be prevented by blocking systemic inflammatory agents. Clinically, only heparin, for inhibition of coagulation phenomena, is presently used among this array of approaches. Corticosteroids have not proven to be beneficial in ARDS. Alternative antiinflammatory agents are being proposed and are under current clinical investigation (e.g. indomethacin, acetylcysteine, alpha 1-proteinase inhibitor, antitumor necrosis factor, interleukin 1 receptor antagonist, platelet-activating factor antagonists). Symptomatic therapeutic strategies in early ARDS include selective pulmonary vasodilation (preferably by inhaled vasorelaxant agents) and optimal fluid balance. Transbronchial surfactant application, presently tested in pilot studies, may be available for ARDS patients in the near future and may have acute beneficial effects on gas exchange, pulmonary mechanics, and lung hemodynamics; its impact on survival cannot be predicted at the present time. Strong efforts should be taken to reduce secondary nosocomial pneumonia in ARDS patients and thus avoid the vicious circle of pneumonia, sepsis from lung infection, and perpetuation of multiple organ dysfunction syndrome. Optimal respirator therapy should be directed to ameliorate gas-exchange conditions acutely but at the same time should aim at minimizing potentially aggravating side effects of artificial ventilation (barotrauma, O2 toxicity). Several new techniques of mechanical ventilation and the concept of permissive hypercapnia address these aspects. Approaches with extracorporeal CO2 removal and oxygenation are being used in specialized centers.

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Hemodynamic Effects of High-Frequency Jet Ventilation

Rouby¹

1987

Update in Intensive Care and Emergency Medicine

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Intermittent Positive Pressure Ventilation with either Positive End-Expiratory Pressure or High Frequency Jet Ventilation (HFJV), or HFJV Alone in Human Acute Respiratory Failure

Cited by 17 publications

References 7 publications

The effect of combined high frequency ventilation with and without continuous positive airway pressure in experimental lung injury

The effect of combined high frequency ventilation with and without continuous positive airway pressure in experimental lung injury

Prevention and therapy of the adult respiratory distress syndrome

Hemodynamic Effects of High-Frequency Jet Ventilation

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