Is intestinal stasis sufficient by itself in promoting enterocolitis in a non-genetic rat model of Hirschsprung’s disease?

Mitroudi, Magdalini; Psalla, Dimitra; Κοντοπούλου, Κωνσταντίνα; Theocharidis, Konstantinos; Sfoungaris, Dimitrios

doi:10.17219/acem/109342

Adv Clin Exp Med

2019

DOI: 10.17219/acem/109342

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Is intestinal stasis sufficient by itself in promoting enterocolitis in a non-genetic rat model of Hirschsprung’s disease?

Magdalini Mitroudi¹,

Dimitra Psalla²,

Κωνσταντίνα Κοντοπούλου³

et al.

Abstract: We would like to thank Prof. Necdet Sut of Trakya University, Turkey, and Erasmus partner, for his valuable help in the statistical analysis of our results.

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Update on the Pathogenesis of the Hirschsprung-Associated Enterocolitis

Zhang

et al. 2023

IJMS

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Despite the significant progress that has been made in terms of understanding the pathophysiology and risk factors of Hirschsprung-associated enterocolitis (HAEC), the morbidity rate has remained unsatisfactorily stable, and clinical management of the condition continues to be challenging. Therefore, in the present literature review, we summarized the up-to-date advances that have been made regarding basic research on the pathogenesis of HAEC. Original articles published between August 2013 and October 2022 were searched in a number of databases, including PubMed, Web of Science, and Scopus. The keywords “Hirschsprung enterocolitis”, “Hirschsprung’s enterocolitis”, “Hirschsprung’s-associated enterocolitis”, and “Hirschsprung-associated enterocolitis” were selected and reviewed. A total of 50 eligible articles were obtained. The latest findings of these research articles were grouped into gene, microbiome, barrier function, enteric nervous system, and immune state categories. The present review concludes that HAEC is shown to be a multifactorial clinical syndrome. Only deep insights into this syndrome, with an accrual of knowledge in terms of understanding its pathogenesis, will elicit the necessary changes that are required for managing this disease.

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Update on the Pathogenesis of the Hirschsprung-Associated Enterocolitis

Zhang

et al. 2023

IJMS

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show abstract

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Is intestinal stasis sufficient by itself in promoting enterocolitis in a non-genetic rat model of Hirschsprung’s disease?

Abstract: We would like to thank Prof. Necdet Sut of Trakya University, Turkey, and Erasmus partner, for his valuable help in the statistical analysis of our results.

Cited by 1 publication

References 30 publications

Update on the Pathogenesis of the Hirschsprung-Associated Enterocolitis

Update on the Pathogenesis of the Hirschsprung-Associated Enterocolitis

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